Matrix Metalloproteinase 9
"Matrix Metalloproteinase 9" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An endopeptidase that is structurally similar to MATRIX METALLOPROTEINASE 2. It degrades GELATIN types I and V; COLLAGEN TYPE IV; and COLLAGEN TYPE V.
| Descriptor ID |
D020780
|
| MeSH Number(s) |
D08.811.277.656.300.480.205.360 D08.811.277.656.300.480.252.445 D08.811.277.656.300.480.525.700.350 D08.811.277.656.675.374.205.360 D08.811.277.656.675.374.252.445 D08.811.277.656.675.374.525.700.350 D12.644.276.848.350 D12.776.467.836.350
|
| Concept/Terms |
Matrix Metalloproteinase 9- Matrix Metalloproteinase 9
- Metalloproteinase 9, Matrix
- 92-kDa Type IV Collagenase
- 92 kDa Type IV Collagenase
- Matrix Metalloproteinase-9
- MMP9 Metalloproteinase
- Metalloproteinase, MMP9
- MMP-9 Metalloproteinase
- MMP 9 Metalloproteinase
- Metalloproteinase, MMP-9
- 92-kDa Gelatinase
- 92 kDa Gelatinase
- Gelatinase B
|
Below are MeSH descriptors whose meaning is more general than "Matrix Metalloproteinase 9".
Below are MeSH descriptors whose meaning is more specific than "Matrix Metalloproteinase 9".
This graph shows the total number of publications written about "Matrix Metalloproteinase 9" by people in this website by year, and whether "Matrix Metalloproteinase 9" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 1997 | 0 | 1 | 1 |
| 1998 | 0 | 1 | 1 |
| 2002 | 0 | 1 | 1 |
| 2003 | 1 | 0 | 1 |
| 2004 | 0 | 1 | 1 |
| 2010 | 0 | 1 | 1 |
| 2011 | 0 | 1 | 1 |
| 2012 | 0 | 2 | 2 |
| 2013 | 3 | 1 | 4 |
| 2014 | 2 | 2 | 4 |
| 2015 | 4 | 4 | 8 |
| 2016 | 1 | 0 | 1 |
| 2017 | 1 | 2 | 3 |
| 2019 | 0 | 1 | 1 |
| 2021 | 1 | 0 | 1 |
| 2022 | 1 | 0 | 1 |
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Below are the most recent publications written about "Matrix Metalloproteinase 9" by people in Profiles.
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Induction of Cellular Senescence in Rat Vaginal Fibroblasts and Treatment With Senolytics: An in Vitro Model for the Study of Pelvic Organ Prolapse. Female Pelvic Med Reconstr Surg. 2022 05 01; 28(5):341-345.
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Effect of genetic depletion of MMP-9 on neurological manifestations of hypertension-induced intracerebral hemorrhages in aged mice. Geroscience. 2021 10; 43(5):2611-2619.
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Exogenous CXCL4 infusion inhibits macrophage phagocytosis by limiting CD36 signalling to enhance post-myocardial infarction cardiac dilation and mortality. Cardiovasc Res. 2019 02 01; 115(2):395-408.
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Inhibition of mTOR protects the blood-brain barrier in models of Alzheimer's disease and vascular cognitive impairment. Am J Physiol Heart Circ Physiol. 2018 04 01; 314(4):H693-H703.
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Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction. Am J Physiol Heart Circ Physiol. 2018 02 01; 314(2):H224-H235.
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Fibroblast growth factor-2 drives and maintains progressive corneal neovascularization following HSV-1 infection. Mucosal Immunol. 2018 01; 11(1):172-185.
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Transgenic overexpression of macrophage matrix metalloproteinase-9 exacerbates age-related cardiac hypertrophy, vessel rarefaction, inflammation, and fibrosis. Am J Physiol Heart Circ Physiol. 2017 Mar 01; 312(3):H375-H383.
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Matrix Metalloproteinase-3 (MMP-3) Is an Endogenous Activator of the MMP-9 Secreted by Placental Leukocytes: Implication in Human Labor. PLoS One. 2015; 10(12):e0145366.
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A Novel Collagen Matricryptin Reduces Left Ventricular Dilation Post-Myocardial Infarction by Promoting Scar Formation and Angiogenesis. J Am Coll Cardiol. 2015 Sep 22; 66(12):1364-74.
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The circular relationship between matrix metalloproteinase-9 and inflammation following myocardial infarction. IUBMB Life. 2015 Aug; 67(8):611-8.