"Herpesvirus 2, Human" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A species of SIMPLEXVIRUS associated with genital infections (HERPES GENITALIS). It is transmitted by sexual intercourse and close personal contact.
|Herpesvirus 2, Human
- Herpesvirus 2, Human
- Herpes Simplex Virus Type 2
- Human Herpesvirus 2
- Herpes Simplex Virus 2
- Herpesvirus 2 (alpha), Human
Below are MeSH descriptors whose meaning is more general than "Herpesvirus 2, Human".
Below are MeSH descriptors whose meaning is more specific than "Herpesvirus 2, Human".
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Below are the most recent publications written about "Herpesvirus 2, Human" by people in Profiles.
Reply to "Highly Efficacious Novel Vaccine, Humoral Immunity, and Ocular Herpes Simplex Virus 1: Reality or Myth?" J Virol. 2017 12 01; 91(23).
Interferon-e protects the female reproductive tract from viral and bacterial infection. Science. 2013 Mar 01; 339(6123):1088-92.
Comparison of the host immune response to herpes simplex virus 1 (HSV-1) and HSV-2 at two different mucosal sites. J Virol. 2012 Jul; 86(13):7454-8.
Vitamin D status and antibody levels to common viruses in pediatric-onset multiple sclerosis. Mult Scler. 2011 Jun; 17(6):666-71.
Loss of the type I interferon pathway increases vulnerability of mice to genital herpes simplex virus 2 infection. J Virol. 2011 Feb; 85(4):1625-33.
CXCR3 deficiency increases susceptibility to genital herpes simplex virus type 2 infection: Uncoupling of CD8+ T-cell effector function but not migration. J Virol. 2009 Sep; 83(18):9486-501.
Herpes simplex virus type 2-induced mortality following genital infection is blocked by anti-tumor necrosis factor alpha antibody in CXCL10-deficient mice. J Virol. 2008 Oct; 82(20):10295-301.
CXCL9 and CXCL10 expression are critical for control of genital herpes simplex virus type 2 infection through mobilization of HSV-specific CTL and NK cells to the nervous system. J Immunol. 2008 Jan 15; 180(2):1098-106.
Susceptibility of CCR5-deficient mice to genital herpes simplex virus type 2 is linked to NK cell mobilization. J Virol. 2007 Apr; 81(8):3704-13.
The lack of RNA-dependent protein kinase enhances susceptibility of mice to genital herpes simplex virus type 2 infection. Immunology. 2006 Aug; 118(4):520-6.