"Herpesvirus 1, Human" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The type species of SIMPLEXVIRUS causing most forms of non-genital herpes simplex in humans. Primary infection occurs mainly in infants and young children and then the virus becomes latent in the dorsal root ganglion. It then is periodically reactivated throughout life causing mostly benign conditions.
| Descriptor ID |
D018259
|
| MeSH Number(s) |
B04.280.382.100.750.390
|
| Concept/Terms |
Herpesvirus 1, Human- Herpesvirus 1, Human
- Herpesvirus 1 (alpha), Human
- Human Herpesvirus 1
- HHV-1
- Herpes Simplex Virus Type 1
- Herpes Simplex Virus 1
- HSV-1
|
Below are MeSH descriptors whose meaning is more general than "Herpesvirus 1, Human".
Below are MeSH descriptors whose meaning is more specific than "Herpesvirus 1, Human".
This graph shows the total number of publications written about "Herpesvirus 1, Human" by people in this website by year, and whether "Herpesvirus 1, Human" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2001 | 0 | 2 | 2 |
| 2002 | 4 | 2 | 6 |
| 2003 | 2 | 1 | 3 |
| 2004 | 1 | 1 | 2 |
| 2005 | 3 | 0 | 3 |
| 2006 | 3 | 2 | 5 |
| 2007 | 2 | 0 | 2 |
| 2008 | 7 | 1 | 8 |
| 2009 | 3 | 0 | 3 |
| 2010 | 0 | 1 | 1 |
| 2011 | 2 | 2 | 4 |
| 2012 | 3 | 1 | 4 |
| 2013 | 1 | 2 | 3 |
| 2014 | 2 | 2 | 4 |
| 2015 | 3 | 0 | 3 |
| 2016 | 4 | 1 | 5 |
| 2017 | 9 | 1 | 10 |
| 2018 | 1 | 1 | 2 |
| 2019 | 1 | 1 | 2 |
| 2020 | 3 | 0 | 3 |
| 2021 | 2 | 1 | 3 |
| 2022 | 4 | 0 | 4 |
| 2023 | 1 | 0 | 1 |
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Below are the most recent publications written about "Herpesvirus 1, Human" by people in Profiles.
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Osteopontin contributes to virus resistance associated with type I IFN expression, activation of downstream ifn-inducible effector genes, and CCR2+CD115+CD206+ macrophage infiltration following ocular HSV-1 infection of mice. Front Immunol. 2022; 13:1028341.
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HSV-1 0?NLS vaccine elicits a robust B lymphocyte response and preserves vision without HSV-1 glycoprotein M or thymidine kinase recognition. Sci Rep. 2022 09 23; 12(1):15920.
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Deletion of the CTRL2 Insulator in HSV-1 Results in the Decreased Expression of Genes Involved in Axonal Transport and Attenuates Reactivation In Vivo. Viruses. 2022 04 27; 14(5).
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Tripartite-Motif 21 (TRIM21) Deficiency Results in a Modest Loss of Herpes Simplex Virus (HSV)-1 Surveillance in the Trigeminal Ganglia Following Cornea Infection. Viruses. 2022 03 12; 14(3).
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Noncognate Signals Drive Enhanced Effector CD8+ T Cell Responses through an IFNAR1-Dependent Pathway after Infection with the Prototypic Vaccine, 0?NLS, against Herpes Simplex Virus 1. J Virol. 2022 03 23; 96(6):e0172421.
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An intact complement system dampens cornea inflammation during acute primary HSV-1 infection. Sci Rep. 2021 05 13; 11(1):10247.
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Lack of neonatal Fc receptor does not diminish the efficacy of the HSV-1 0?NLS vaccine against ocular HSV-1 challenge. Vaccine. 2021 04 28; 39(18):2526-2536.
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Herpes Simplex Virus 1 MicroRNA miR-H8 Is Dispensable for Latency and Reactivation In Vivo. J Virol. 2021 01 28; 95(4).
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Herpes Simplex Virus 1 (HSV-1) 0?NLS Live-Attenuated Vaccine Protects against Ocular HSV-1 Infection in the Absence of Neutralizing Antibody in HSV-1 gB T Cell Receptor-Specific Transgenic Mice. J Virol. 2020 11 23; 94(24).
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Distinguishing Features of High- and Low-Dose Vaccine against Ocular HSV-1 Infection Correlates with Recognition of Specific HSV-1-Encoded Proteins. Immunohorizons. 2020 10 09; 4(10):608-626.