"Iron-Binding Proteins" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Proteins that specifically bind to IRON.
Descriptor ID |
D033862
|
MeSH Number(s) |
D12.776.157.427 D12.776.556.579
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Concept/Terms |
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Below are MeSH descriptors whose meaning is more general than "Iron-Binding Proteins".
Below are MeSH descriptors whose meaning is more specific than "Iron-Binding Proteins".
This graph shows the total number of publications written about "Iron-Binding Proteins" by people in this website by year, and whether "Iron-Binding Proteins" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
Year | Major Topic | Minor Topic | Total |
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1996 | 1 | 0 | 1 |
1997 | 2 | 2 | 4 |
1998 | 2 | 1 | 3 |
1999 | 1 | 0 | 1 |
2002 | 0 | 1 | 1 |
2004 | 3 | 0 | 3 |
2006 | 1 | 2 | 3 |
2007 | 3 | 1 | 4 |
2009 | 1 | 0 | 1 |
2014 | 2 | 0 | 2 |
2015 | 1 | 0 | 1 |
2016 | 1 | 1 | 2 |
2019 | 1 | 0 | 1 |
2022 | 0 | 1 | 1 |
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Below are the most recent publications written about "Iron-Binding Proteins" by people in Profiles.
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DNA methylation in Friedreich ataxia silences expression of frataxin isoform E. Sci Rep. 2022 03 23; 12(1):5031.
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Quantitative Proteomics of Presynaptic Mitochondria Reveal an Overexpression and Biological Relevance of Neuronal MitoNEET in Postnatal Brain Development. Dev Neurobiol. 2019 04; 79(4):370-386.
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Congenital hypothyroidism and thyroid dyshormonogenesis: a case report of siblings with a newly identified mutation in thyroperoxidase. J Pediatr Endocrinol Metab. 2016 May 01; 29(5):627-31.
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Reversal of epigenetic promoter silencing in Friedreich ataxia by a class I histone deacetylase inhibitor. Nucleic Acids Res. 2016 06 20; 44(11):5095-104.
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FXN Promoter Silencing in the Humanized Mouse Model of Friedreich Ataxia. PLoS One. 2015; 10(9):e0138437.
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Epigenetic promoter silencing in Friedreich ataxia is dependent on repeat length. Ann Neurol. 2014 Oct; 76(4):522-8.
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Altered nucleosome positioning at the transcription start site and deficient transcriptional initiation in Friedreich ataxia. J Biol Chem. 2014 May 30; 289(22):15194-202.
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Epigenetic silencing in Friedreich ataxia is associated with depletion of CTCF (CCCTC-binding factor) and antisense transcription. PLoS One. 2009 Nov 19; 4(11):e7914.
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Repair of DNA double-strand breaks within the (GAA*TTC)n sequence results in frequent deletion of the triplet-repeat sequence. Nucleic Acids Res. 2008 Feb; 36(2):489-500.
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Deficiency of RecA-dependent RecFOR and RecBCD pathways causes increased instability of the (GAA*TTC)n sequence when GAA is the lagging strand template. Nucleic Acids Res. 2007; 35(20):6884-94.