"Calgranulin B" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A 13.2-kDa member of the S-100 family of calcium-binding proteins that can form homo- or heterocomplexes with CALGRANULIN A and a variety of other proteins. The calgranulin A/B heterodimer is known as LEUKOCYTE L1 ANTIGEN COMPLEX. Calgranulin B is expressed at high concentrations in GRANULOCYTES during early monocyte differentiation, and serum calgranulin B levels are elevated in many inflammatory disorders such as CYSTIC FIBROSIS.
| Descriptor ID |
D040502
|
| MeSH Number(s) |
D12.776.157.125.750.500.200 D12.776.631.655.500.200 D23.050.301.562.200
|
| Concept/Terms |
Calgranulin B- Calgranulin B
- CABP-P14
- MRP14 Protein
- Leukocyte L1 Protein Heavy Chain
- Antigen 60B8-A
- Antigen 60B8 A
- Macrophage-Related Protein-14
- Macrophage Related Protein 14
- Myeloid Calcium-Binding Protein p14
- Myeloid Calcium Binding Protein p14
- MRP-14 Protein
- MRP 14 Protein
- 60B8-A Antigen
- 60B8 A Antigen
- Antigen, 60B8-A
- S100A9 Protein
|
Below are MeSH descriptors whose meaning is more general than "Calgranulin B".
Below are MeSH descriptors whose meaning is more specific than "Calgranulin B".
This graph shows the total number of publications written about "Calgranulin B" by people in this website by year, and whether "Calgranulin B" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2012 | 0 | 1 | 1 |
| 2014 | 0 | 1 | 1 |
| 2017 | 1 | 0 | 1 |
| 2019 | 1 | 0 | 1 |
| 2020 | 3 | 2 | 5 |
| 2021 | 1 | 0 | 1 |
| 2022 | 0 | 1 | 1 |
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Below are the most recent publications written about "Calgranulin B" by people in Profiles.
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Targeting Doublecortin-Like Kinase 1 (DCLK1)-Regulated SARS-CoV-2 Pathogenesis in COVID-19. J Virol. 2022 09 14; 96(17):e0096722.
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DAMPening Mortality in COVID-19: Therapeutic Insights From Basic Cardiometabolic Studies on S100A8/A9. Circulation. 2021 03 09; 143(10):971-973.
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Emerging roles of neutrophil-borne S100A8/A9 in cardiovascular inflammation. Pharmacol Res. 2020 11; 161:105212.
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NETosis Is Required for S100A8/A9-Induced Granulopoiesis After Myocardial Infarction. Arterioscler Thromb Vasc Biol. 2020 11; 40(11):2805-2807.
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Response by Sreejit and Nagareddy to Letter Regarding Article, "Neutrophil-Derived S100A8/A9 Amplify Granulopoiesis After Myocardial Infarction". Circulation. 2020 09; 142(9):e125-e126.
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Transient Intermittent Hyperglycemia Accelerates Atherosclerosis by Promoting Myelopoiesis. Circ Res. 2020 09 11; 127(7):877-892.
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Antimicrobial Peptides Derived from the Immune Defense Protein CAP37 Inhibit TLR4 Activation by S100A9. Invest Ophthalmol Vis Sci. 2020 04 09; 61(4):16.
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S100A8/A9 in Myocardial Infarction. Methods Mol Biol. 2019; 1929:739-754.
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Neutrophil-derived S100 calcium-binding proteins A8/A9 promote reticulated thrombocytosis and atherogenesis in diabetes. J Clin Invest. 2017 Jun 01; 127(6):2133-2147.
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Targeting of BRAF resistant melanoma via extracellular matrix metalloproteinase inducer receptor. J Surg Res. 2014 Jul; 190(1):111-8.