Transcription Factor RelA
"Transcription Factor RelA" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A subunit of NF-kappa B that is primarily responsible for its transactivation function. It contains a C-terminal transactivation domain and an N-terminal domain with homology to PROTO-ONCOGENE PROTEINS C-REL.
|Transcription Factor RelA
- Transcription Factor RelA
- RelA, Transcription Factor
- NF-kappa B p65
- NF kappa B p65
- p65, NF-kappa B
- NF-kappa B p65 Subunit
- NF kappa B p65 Subunit
- Transcription Factor p65
- p65, Transcription Factor
- p65 Transcription Factor
- Transcription Factor, p65
- v-Rel Reticuloendotheliosis Viral Oncogene Homolog A
- v Rel Reticuloendotheliosis Viral Oncogene Homolog A
- NF-kappaB 65-kDa
- NF kappaB 65 kDa
Below are MeSH descriptors whose meaning is more general than "Transcription Factor RelA".
Below are MeSH descriptors whose meaning is more specific than "Transcription Factor RelA".
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Below are the most recent publications written about "Transcription Factor RelA" by people in Profiles.
An incoherent feedforward loop interprets NF?B/RelA dynamics to determine TNF-induced necroptosis decisions. Mol Syst Biol. 2020 12; 16(12):e9677.
Targeted analysis of recombinant NF kappa B (RelA/p65) by denaturing and native top down mass spectrometry. J Proteomics. 2016 Feb 16; 134:76-84.
17ß estradiol regulates adhesion molecule expression in mesangial cells during glomerulonephritis. Clin Immunol. 2015 Jul; 159(1):13-22.
Metastatic neuroblastoma cancer stem cells exhibit flexible plasticity and adaptive stemness signaling. Stem Cell Res Ther. 2015 Feb 20; 6:2.
Acquired tumor cell radiation resistance at the treatment site is mediated through radiation-orchestrated intercellular communication. Int J Radiat Oncol Biol Phys. 2014 Mar 01; 88(3):677-85.
Preclinical evaluation of 4-[3,5-bis(2-chlorobenzylidene)-4-oxo-piperidine-1-yl]-4-oxo-2-butenoic acid, in a mouse model of lung cancer xenograft. Br J Pharmacol. 2013 Dec; 170(7):1436-48.
Chitosan oligosaccharides block LPS-induced O-GlcNAcylation of NF-?B and endothelial inflammatory response. Carbohydr Polym. 2014 Jan; 99:568-78.
Pharmacologic suppression of inflammation by a diphenyldifluoroketone, EF24, in a rat model of fixed-volume hemorrhage improves survival. J Pharmacol Exp Ther. 2013 Nov; 347(2):346-56.
Molecular basis of 'hypoxic' breast cancer cell radio-sensitization: phytochemicals converge on radiation induced Rel signaling. Radiat Oncol. 2013 Mar 04; 8:46.
CCAAT/enhancer-binding protein ß (C/EBPß) expression regulates dietary-induced inflammation in macrophages and adipose tissue in mice. J Biol Chem. 2012 Oct 05; 287(41):34349-60.