Nitric Oxide Synthase Type III
"Nitric Oxide Synthase Type III" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A CALCIUM-dependent, constitutively-expressed form of nitric oxide synthase found primarily in ENDOTHELIAL CELLS.
| Descriptor ID |
D052250
|
| MeSH Number(s) |
D08.811.682.664.500.772.750
|
| Concept/Terms |
Nitric Oxide Synthase Type III- Nitric Oxide Synthase Type III
- Nitric Oxide Synthase, Type III
- Endothelial Nitric Oxide Synthase
- ENOS Enzyme
- ECNOS Enzyme
- Endothelial Constitutive Nitric Oxide Synthase
|
Below are MeSH descriptors whose meaning is more general than "Nitric Oxide Synthase Type III".
Below are MeSH descriptors whose meaning is more specific than "Nitric Oxide Synthase Type III".
This graph shows the total number of publications written about "Nitric Oxide Synthase Type III" by people in this website by year, and whether "Nitric Oxide Synthase Type III" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2000 | 0 | 1 | 1 |
| 2001 | 0 | 1 | 1 |
| 2002 | 0 | 1 | 1 |
| 2005 | 1 | 2 | 3 |
| 2006 | 1 | 4 | 5 |
| 2007 | 1 | 1 | 2 |
| 2008 | 1 | 1 | 2 |
| 2009 | 1 | 6 | 7 |
| 2010 | 0 | 2 | 2 |
| 2011 | 1 | 0 | 1 |
| 2012 | 0 | 2 | 2 |
| 2013 | 1 | 1 | 2 |
| 2014 | 0 | 2 | 2 |
| 2015 | 2 | 0 | 2 |
| 2016 | 0 | 1 | 1 |
| 2017 | 1 | 2 | 3 |
| 2021 | 1 | 1 | 2 |
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Below are the most recent publications written about "Nitric Oxide Synthase Type III" by people in Profiles.
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Association of Endothelial Nitric Oxide Synthase Gene Polymorphisms with Coronary Artery Disease in North Indian Punjabi Population. Biochem Genet. 2022 Dec; 60(6):2120-2136.
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Preserved ß-adrenergic-mediated vasodilation in skeletal muscle of young adults with obesity despite shifts in cyclooxygenase and nitric oxide synthase. Am J Physiol Heart Circ Physiol. 2022 01 01; 322(1):H25-H35.
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mTOR Attenuation with Rapamycin Reverses Neurovascular Uncoupling and Memory Deficits in Mice Modeling Alzheimer's Disease. J Neurosci. 2021 05 12; 41(19):4305-4320.
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Phenotypic Consequences of a Genetic Predisposition to Enhanced Nitric Oxide Signaling. Circulation. 2018 01 16; 137(3):222-232.
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Conditional Deletion of Bmal1 Accentuates Microvascular and Macrovascular Injury. Am J Pathol. 2017 Jun; 187(6):1426-1435.
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Inflammation-induced caveolin-1 and BMPRII depletion promotes endothelial dysfunction and TGF-ß-driven pulmonary vascular remodeling. Am J Physiol Lung Cell Mol Physiol. 2017 05 01; 312(5):L760-L771.
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Monocrotaline-Induced Pulmonary Hypertension Involves Downregulation of Antiaging Protein Klotho and eNOS Activity. Hypertension. 2016 11; 68(5):1255-1263.
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Resveratrol supplementation confers neuroprotection in cortical brain tissue of nonhuman primates fed a high-fat/sucrose diet. Aging (Albany NY). 2016 05; 8(5):899-916.
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Purinergic glio-endothelial coupling during neuronal activity: role of P2Y1 receptors and eNOS in functional hyperemia in the mouse somatosensory cortex. Am J Physiol Heart Circ Physiol. 2015 Dec 01; 309(11):H1837-45.
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Adrenocorticotropic Hormone and PI3K/Akt Inhibition Reduce eNOS Phosphorylation and Increase Cortisol Biosynthesis in Long-Term Hypoxic Ovine Fetal Adrenal Cortical Cells. Reprod Sci. 2015 Aug; 22(8):932-41.