NAV1.8 Voltage-Gated Sodium Channel
"NAV1.8 Voltage-Gated Sodium Channel" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A voltage-gated sodium channel subtype that is expressed in nociceptors, including spinal and trigeminal sensory neurons. It plays a role in the transmission of pain signals induced by cold, heat, and mechanical stimuli.
Descriptor ID |
D062559
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MeSH Number(s) |
D12.776.157.530.400.875.750.800 D12.776.543.550.450.875.750.800 D12.776.543.585.400.875.750.800 D12.776.631.960.800
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Concept/Terms |
NAV1.8 Voltage-Gated Sodium Channel- NAV1.8 Voltage-Gated Sodium Channel
- NAV1.8 Voltage Gated Sodium Channel
- Peripheral Nerve Sodium Channel 3
- Sodium Channel Protein Type X
- PN3 Sodium Channel
- Sodium Channel, PN3
- Type 10 Voltage-Gated Sodium Channel
- Type 10 Voltage Gated Sodium Channel
- Voltage-Gated Sodium Channel Type 10
- Voltage Gated Sodium Channel Type 10
Sodium Channel Protein Type 10 Subunit alpha- Sodium Channel Protein Type 10 Subunit alpha
- SCN10A Sodium Channel alpha Subunit
- Voltage-Gated Sodium Channel Type 10 Subunit alpha
- Voltage Gated Sodium Channel Type 10 Subunit alpha
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Below are MeSH descriptors whose meaning is more general than "NAV1.8 Voltage-Gated Sodium Channel".
Below are MeSH descriptors whose meaning is more specific than "NAV1.8 Voltage-Gated Sodium Channel".
This graph shows the total number of publications written about "NAV1.8 Voltage-Gated Sodium Channel" by people in this website by year, and whether "NAV1.8 Voltage-Gated Sodium Channel" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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2006 | 0 | 2 | 2 |
2021 | 1 | 0 | 1 |
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Below are the most recent publications written about "NAV1.8 Voltage-Gated Sodium Channel" by people in Profiles.
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Identification and Characterization of Novel Proteins from Arizona Bark Scorpion Venom That Inhibit Nav1.8, a Voltage-Gated Sodium Channel Regulator of Pain Signaling. Toxins (Basel). 2021 07 18; 13(7).
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Analysis of the variation in use-dependent inactivation of high-threshold tetrodotoxin-resistant sodium currents recorded from rat sensory neurons. Neuroscience. 2006 Dec 28; 143(4):923-38.
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Carbamazepine interacts with a slow inactivation state of NaV1.8-like sodium channels. Neurosci Lett. 2006 Nov 13; 408(2):129-34.