"Nociceptors" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Peripheral AFFERENT NEURONS which are sensitive to injuries or pain, usually caused by extreme thermal exposures, mechanical forces, or other noxious stimuli. Their cell bodies reside in the DORSAL ROOT GANGLIA. Their peripheral terminals (NERVE ENDINGS) innervate target tissues and transduce noxious stimuli via axons to the CENTRAL NERVOUS SYSTEM.
Descriptor ID |
D009619
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MeSH Number(s) |
A08.675.650.915.875 A08.800.950.875 A11.671.650.915.875
|
Concept/Terms |
Nociceptors- Nociceptors
- Nociceptor
- Pain Receptors
- Pain Receptor
- Receptors, Pain
- Nociceptive Neurons
- Neuron, Nociceptive
- Neurons, Nociceptive
- Nociceptive Neuron
|
Below are MeSH descriptors whose meaning is more general than "Nociceptors".
Below are MeSH descriptors whose meaning is more specific than "Nociceptors".
This graph shows the total number of publications written about "Nociceptors" by people in this website by year, and whether "Nociceptors" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
Year | Major Topic | Minor Topic | Total |
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1998 | 0 | 1 | 1 |
1999 | 0 | 1 | 1 |
2000 | 0 | 1 | 1 |
2001 | 1 | 0 | 1 |
2003 | 1 | 0 | 1 |
2004 | 1 | 1 | 2 |
2006 | 3 | 3 | 6 |
2007 | 3 | 3 | 6 |
2008 | 2 | 1 | 3 |
2009 | 2 | 0 | 2 |
2010 | 1 | 4 | 5 |
2011 | 2 | 1 | 3 |
2012 | 2 | 1 | 3 |
2014 | 0 | 1 | 1 |
2016 | 1 | 0 | 1 |
2019 | 0 | 1 | 1 |
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Below are the most recent publications written about "Nociceptors" by people in Profiles.
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Race/Ethnicity Does Not Moderate the Relationship Between Adverse Life Experiences and Temporal Summation of the Nociceptive Flexion Reflex and Pain: Results From the Oklahoma Study of Native American Pain Risk. J Pain. 2019 08; 20(8):941-955.
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Behavioral Inhibition and Behavioral Activation are Related to Habituation of Nociceptive Flexion Reflex, but Not Pain Ratings. J Pain. 2017 03; 18(3):349-358.
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Stress-induced pain: a target for the development of novel therapeutics. J Pharmacol Exp Ther. 2014 Nov; 351(2):327-35.
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Using multilevel growth curve modeling to examine emotional modulation of temporal summation of pain (TS-pain) and the nociceptive flexion reflex (TS-NFR). Pain. 2012 Nov; 153(11):2274-2282.
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Endogenous inhibition of the nociceptive flexion reflex (NFR) and pain ratings during the menstrual cycle in healthy women. Ann Behav Med. 2012 Jun; 43(3):343-51.
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Is constant current or constant voltage spinal cord stimulation superior for the suppression of nociceptive visceral and somatic stimuli? A rat model. Neuromodulation. 2012 Mar-Apr; 15(2):132-42; discussion 143.
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Standardizing procedures to study sensitization of human spinal nociceptive processes: comparing parameters for temporal summation of the nociceptive flexion reflex (TS-NFR). Int J Psychophysiol. 2011 Sep; 81(3):263-74.
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Pain catastrophizing is related to temporal summation of pain but not temporal summation of the nociceptive flexion reflex. Pain. 2011 Apr; 152(4):794-801.
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Serotonin transporter gene (5-HTTLPR) polymorphisms are associated with emotional modulation of pain but not emotional modulation of spinal nociception. Biol Psychol. 2011 Mar; 86(3):360-9.
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Upper thoracic postsynaptic dorsal column neurons conduct cardiac mechanoreceptive information, but not cardiac chemical nociception in rats. Brain Res. 2010 Dec 17; 1366:71-84.