"Enterococcus faecalis" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A species of gram-positive, coccoid bacteria commonly isolated from clinical specimens and the human intestinal tract. Most strains are nonhemolytic.
| Descriptor ID |
D013293
|
| MeSH Number(s) |
B03.353.750.250.250.280 B03.510.550.250.250.280
|
| Concept/Terms |
|
Below are MeSH descriptors whose meaning is more general than "Enterococcus faecalis".
Below are MeSH descriptors whose meaning is more specific than "Enterococcus faecalis".
This graph shows the total number of publications written about "Enterococcus faecalis" by people in this website by year, and whether "Enterococcus faecalis" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1995 | 3 | 0 | 3 |
| 1996 | 1 | 0 | 1 |
| 1997 | 1 | 1 | 2 |
| 1998 | 1 | 1 | 2 |
| 1999 | 2 | 0 | 2 |
| 2001 | 2 | 0 | 2 |
| 2002 | 5 | 0 | 5 |
| 2003 | 1 | 0 | 1 |
| 2004 | 4 | 0 | 4 |
| 2005 | 4 | 0 | 4 |
| 2006 | 4 | 0 | 4 |
| 2007 | 1 | 1 | 2 |
| 2008 | 3 | 0 | 3 |
| 2009 | 1 | 0 | 1 |
| 2010 | 1 | 0 | 1 |
| 2011 | 1 | 0 | 1 |
| 2012 | 2 | 1 | 3 |
| 2013 | 1 | 0 | 1 |
| 2014 | 3 | 1 | 4 |
| 2015 | 2 | 0 | 2 |
| 2016 | 1 | 0 | 1 |
| 2019 | 0 | 1 | 1 |
To return to the timeline,
click here.
Below are the most recent publications written about "Enterococcus faecalis" by people in Profiles.
-
Risks associated with enterococci as probiotics. Food Res Int. 2020 03; 129:108788.
-
The opportunistic pathogen Enterococcus faecalis resists phagosome acidification and autophagy to promote intracellular survival in macrophages. Cell Microbiol. 2016 06; 18(6):831-43.
-
Surface protein Esp enhances pro-inflammatory cytokine expression through NF-?B activation during enterococcal infection. Innate Immun. 2016 Jan; 22(1):31-9.
-
Roles of TLR/MyD88/MAPK/NF-?B Signaling Pathways in the Regulation of Phagocytosis and Proinflammatory Cytokine Expression in Response to E. faecalis Infection. PLoS One. 2015; 10(8):e0136947.
-
A TIR domain protein from E. faecalis attenuates MyD88-mediated signaling and NF-?B activation. PLoS One. 2014; 9(11):e112010.
-
Enterococcus faecalis infection activates phosphatidylinositol 3-kinase signaling to block apoptotic cell death in macrophages. Infect Immun. 2014 Dec; 82(12):5132-42.
-
Commensal bacteria drive endogenous transformation and tumour stem cell marker expression through a bystander effect. Gut. 2015 Mar; 64(3):459-68.
-
The structure and competitive substrate inhibition of dihydrofolate reductase from Enterococcus faecalis reveal restrictions to cofactor docking. Biochemistry. 2014 Feb 25; 53(7):1228-38.
-
Cyclooxygenase-2 generates the endogenous mutagen trans-4-hydroxy-2-nonenal in Enterococcus faecalis-infected macrophages. Cancer Prev Res (Phila). 2013 Mar; 6(3):206-16.
-
TNF-a mediates macrophage-induced bystander effects through Netrin-1. Cancer Res. 2012 Oct 15; 72(20):5219-29.