"Receptors, Interferon" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Specific molecular sites or structures on or in cells with which interferons react or to which they bind in order to modify the function of the cells. Interferons exert their pleiotropic effects through two different receptors. alpha- and beta-interferon crossreact with common receptors, while gamma-interferon initiates its biological effects through its own specific receptor system.
Descriptor ID |
D017471
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MeSH Number(s) |
D12.776.543.750.705.852.400
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Concept/Terms |
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Below are MeSH descriptors whose meaning is more general than "Receptors, Interferon".
Below are MeSH descriptors whose meaning is more specific than "Receptors, Interferon".
This graph shows the total number of publications written about "Receptors, Interferon" by people in this website by year, and whether "Receptors, Interferon" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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2004 | 0 | 1 | 1 |
2006 | 0 | 1 | 1 |
2007 | 0 | 1 | 1 |
2009 | 0 | 1 | 1 |
2021 | 1 | 1 | 2 |
2023 | 1 | 0 | 1 |
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Below are the most recent publications written about "Receptors, Interferon" by people in Profiles.
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Tetraspanin Tspan8 restrains interferon signaling to stabilize intestinal epithelium by directing endocytosis of interferon receptor. Cell Mol Life Sci. 2023 May 19; 80(6):154.
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Genetic Association of a Gain-of-Function IFNGR1 Polymorphism and the Intergenic Region LNCAROD/DKK1 With Behçet's Disease. Arthritis Rheumatol. 2021 07; 73(7):1244-1252.
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Chromatin Looping Links Target Genes with Genetic Risk Loci for Dermatological Traits. J Invest Dermatol. 2021 08; 141(8):1975-1984.
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Plasma from systemic lupus patients compromises cholesterol homeostasis: a potential mechanism linking autoimmunity to atherosclerotic cardiovascular disease. Rheumatol Int. 2010 Mar; 30(5):591-8.
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Oligoadenylate synthetase/protein kinase R pathways and alphabeta TCR+ T cells are required for adenovirus vector: IFN-gamma inhibition of herpes simplex virus-1 in cornea. J Immunol. 2007 Apr 15; 178(8):5166-72.
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OAS and PKR are not required for the antiviral effect of Ad:IFN-gamma against acute HSV-1 in primary trigeminal ganglia cultures. J Interferon Cytokine Res. 2006 Apr; 26(4):220-5.
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Loss of interferon-induced Stat1 phosphorylation in activated T cells. J Interferon Cytokine Res. 2004 Mar; 24(3):169-78.