Prenatal Exposure Delayed Effects
"Prenatal Exposure Delayed Effects" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The consequences of exposing the FETUS in utero to certain factors, such as NUTRITION PHYSIOLOGICAL PHENOMENA; PHYSIOLOGICAL STRESS; DRUGS; RADIATION; and other physical or chemical factors. These consequences are observed later in the offspring after BIRTH.
| Descriptor ID |
D011297
|
| MeSH Number(s) |
C13.703.824.500
|
| Concept/Terms |
|
Below are MeSH descriptors whose meaning is more general than "Prenatal Exposure Delayed Effects".
Below are MeSH descriptors whose meaning is more specific than "Prenatal Exposure Delayed Effects".
This graph shows the total number of publications written about "Prenatal Exposure Delayed Effects" by people in this website by year, and whether "Prenatal Exposure Delayed Effects" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 1 | 0 | 1 |
| 1997 | 1 | 2 | 3 |
| 1998 | 0 | 1 | 1 |
| 2001 | 0 | 1 | 1 |
| 2002 | 1 | 0 | 1 |
| 2004 | 2 | 0 | 2 |
| 2005 | 1 | 0 | 1 |
| 2008 | 1 | 0 | 1 |
| 2010 | 1 | 1 | 2 |
| 2011 | 2 | 2 | 4 |
| 2012 | 3 | 0 | 3 |
| 2013 | 1 | 2 | 3 |
| 2014 | 1 | 2 | 3 |
| 2015 | 1 | 1 | 2 |
| 2016 | 4 | 1 | 5 |
| 2017 | 1 | 1 | 2 |
| 2018 | 6 | 4 | 10 |
| 2019 | 1 | 2 | 3 |
| 2020 | 3 | 3 | 6 |
| 2021 | 2 | 0 | 2 |
| 2023 | 1 | 0 | 1 |
| 2024 | 1 | 1 | 2 |
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Below are the most recent publications written about "Prenatal Exposure Delayed Effects" by people in Profiles.
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Vertical Transfer of Maternal Gut Microbes to Offspring of Western Diet-Fed Dams Drives Reduced Levels of Tryptophan Metabolites and Postnatal Innate Immune Response. Nutrients. 2024 Jun 08; 16(12).
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Developmental Programming of the Fetal Immune System by Maternal Western-Style Diet: Mechanisms and Implications for Disease Pathways in the Offspring. Int J Mol Sci. 2024 May 29; 25(11).
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Exposure-response relationships for personal exposure to fine particulate matter (PM2?5), carbon monoxide, and black carbon and birthweight: an observational analysis of the multicountry Household Air Pollution Intervention Network (HAPIN) trial. Lancet Planet Health. 2023 05; 7(5):e387-e396.
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Effects of early daily alcohol exposure on placental function and fetal growth in a rhesus macaque model. Am J Obstet Gynecol. 2022 01; 226(1):130.e1-130.e11.
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Reply to Li et al.: Estimate of the association between TB risk and famine intensity is robust to various famine intensity estimators. Proc Natl Acad Sci U S A. 2021 04 20; 118(16).
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Gestational Diabetes Is Uniquely Associated With Altered Early Seeding of the Infant Gut Microbiota. Front Endocrinol (Lausanne). 2020; 11:603021.
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Prenatal and early-life exposure to the Great Chinese Famine increased the risk of tuberculosis in adulthood across two generations. Proc Natl Acad Sci U S A. 2020 11 03; 117(44):27549-27555.
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Type 2 Diabetes in Youth: the Role of Early Life Exposures. Curr Diab Rep. 2020 08 07; 20(9):45.
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Fetal circulating human resistin increases in diabetes during pregnancy and impairs placental mitochondrial biogenesis. Mol Med. 2020 08 06; 26(1):76.
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Prevalence of NAFLD in Guatemala following exposure to a protein-energy nutrition intervention in early life. Ann Hepatol. 2020 Jul - Aug; 19(4):373-379.