Transcription Factor CHOP
"Transcription Factor CHOP" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A CCAAT-enhancer binding protein that is induced by DNA DAMAGE and growth arrest. It serves as a dominant negative inhibitor of other CCAAT-enhancer binding proteins.
| Descriptor ID |
D051742
|
| MeSH Number(s) |
D12.776.260.108.124.875 D12.776.660.167.875 D12.776.930.127.124.875
|
| Concept/Terms |
Transcription Factor CHOP- Transcription Factor CHOP
- CHOP, Transcription Factor
- Growth Arrest And DNA Damage-Inducible Protein 153
- Growth Arrest And DNA Damage Inducible Protein 153
- DNA Damage-Inducible Transcript 3 Protein
- DNA Damage Inducible Transcript 3 Protein
- GADD153 Protein
- C-EBP Homologous Protein
- C EBP Homologous Protein
- Homologous Protein, C-EBP
- DDIT3 Protein
|
Below are MeSH descriptors whose meaning is more general than "Transcription Factor CHOP".
Below are MeSH descriptors whose meaning is more specific than "Transcription Factor CHOP".
This graph shows the total number of publications written about "Transcription Factor CHOP" by people in this website by year, and whether "Transcription Factor CHOP" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2007 | 0 | 2 | 2 |
| 2008 | 1 | 1 | 2 |
| 2015 | 0 | 1 | 1 |
| 2016 | 1 | 0 | 1 |
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Below are the most recent publications written about "Transcription Factor CHOP" by people in Profiles.
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Identification of 1,2,3-triazole derivatives that protect pancreatic ß cells against endoplasmic reticulum stress-mediated dysfunction and death through the inhibition of C/EBP-homologous protein expression. Bioorg Med Chem. 2016 06 15; 24(12):2621-30.
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Resveratrol triggers ER stress-mediated apoptosis by disrupting N-linked glycosylation of proteins in ovarian cancer cells. Cancer Lett. 2016 Feb 28; 371(2):347-53.
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Critical role of proteostasis-imbalance in pathogenesis of COPD and severe emphysema. J Mol Med (Berl). 2011 Jun; 89(6):577-93.
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p21(Cip1) protects against oxidative stress by suppressing ER-dependent activation of mitochondrial death pathways. Free Radic Biol Med. 2009 Jan 01; 46(1):33-41.
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CAAT/enhancer binding protein homologous protein-dependent death receptor 5 induction is a major component of SHetA2-induced apoptosis in lung cancer cells. Cancer Res. 2008 Jul 01; 68(13):5335-44.
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Expression of hereditary hemochromatosis C282Y HFE protein in HEK293 cells activates specific endoplasmic reticulum stress responses. BMC Cell Biol. 2007 Jul 24; 8:30.
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CCAAT/enhancing binding protein beta deletion in mice attenuates inflammation, endoplasmic reticulum stress, and lipid accumulation in diet-induced nonalcoholic steatohepatitis. Hepatology. 2007 May; 45(5):1108-17.
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Foetal rat lung epithelial (FRLE) cells: alterations in cellular homeostasis and gene expression in response to etoposide, hydrogen peroxide and sodium butyrate. Toxicology. 2004 Feb 15; 195(2-3):209-20.