Home
About
Overview
Sharing Data
ORCID
Help
History (2)
Compliance on Mandatory Data Reporting in Registered Obstetrics Trials.
Inflammation-induced caveolin-1 and BMPRII depletion promotes endothelial dysfunction and TGF-ß-driven pulmonary vascular remodeling.
See All 2 Pages
Find People
Find Everything
Login
to edit your profile (add a photo, awards, links to other websites, etc.)
Edit My Profile
My Person List (
0
)
Return to Top
Inflammation-induced caveolin-1 and BMPRII depletion promotes endothelial dysfunction and TGF-ß-driven pulmonary vascular remodeling.
Inflammation-induced caveolin-1 and BMPRII depletion promotes endothelial dysfunction and TGF-ß-driven pulmonary vascular remodeling. Am J Physiol Lung Cell Mol Physiol. 2017 05 01; 312(5):L760-L771.
View in:
PubMed
subject areas
Actins
Actins
Acute Lung Injury
Acute Lung Injury
Adult
Adult
Aged
Aged
Animals
Animals
Bone Morphogenetic Protein Receptors, Type II
Bone Morphogenetic Protein Receptors, Type II
Bronchoalveolar Lavage Fluid
Bronchoalveolar Lavage Fluid
Caveolin 1
Caveolin 1
Cell Shape
Cell Shape
Cytokines
Cytokines
Endothelial Cells
Endothelial Cells
Female
Female
Humans
Humans
Inflammation
Inflammation
Inflammation Mediators
Inflammation Mediators
Interleukin-6
Interleukin-6
Lipopolysaccharides
Lipopolysaccharides
Lung
Lung
Male
Male
Mice, Inbred C57BL
Mice, Inbred C57BL
Middle Aged
Middle Aged
Models, Biological
Models, Biological
Nitric Oxide Synthase Type III
Nitric Oxide Synthase Type III
Proteolysis
Proteolysis
Pulmonary Artery
Pulmonary Artery
Receptors, Transforming Growth Factor beta
Receptors, Transforming Growth Factor beta
Time Factors
Time Factors
Transforming Growth Factor beta
Transforming Growth Factor beta
Vascular Remodeling
Vascular Remodeling
authors with profiles
Michael Hale Elliott