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Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction.
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Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction.
Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction. Am J Physiol Heart Circ Physiol. 2018 02 01; 314(2):H224-H235.
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PubMed
subject areas
Age Factors
Age Factors
Aging
Aging
Animals
Animals
Collagen
Collagen
Diastole
Diastole
Disease Models, Animal
Disease Models, Animal
Enzyme Induction
Enzyme Induction
Female
Female
Fibroblasts
Fibroblasts
Fibrosis
Fibrosis
Inflammation Mediators
Inflammation Mediators
Macrophages
Macrophages
Male
Male
Matrix Metalloproteinase 9
Matrix Metalloproteinase 9
Mice, Inbred C57BL
Mice, Inbred C57BL
Mice, Transgenic
Mice, Transgenic
Myocardial Infarction
Myocardial Infarction
Myocardium
Myocardium
Neovascularization, Physiologic
Neovascularization, Physiologic
Phenotype
Phenotype
Ventricular Function, Left
Ventricular Function, Left
Ventricular Remodeling
Ventricular Remodeling
Wound Healing
Wound Healing
authors with profiles
Andriy Yabluchanskiy