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AKR1C2 and AKR1C3 mediated prostaglandin D2 metabolism augments the PI3K/Akt proliferative signaling pathway in human prostate cancer cells.
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AKR1C2 and AKR1C3 mediated prostaglandin D2 metabolism augments the PI3K/Akt proliferative signaling pathway in human prostate cancer cells.
AKR1C2 and AKR1C3 mediated prostaglandin D2 metabolism augments the PI3K/Akt proliferative signaling pathway in human prostate cancer cells. Mol Cell Endocrinol. 2008 Jul 16; 289(1-2):60-6.
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PubMed
subject areas
3-Hydroxysteroid Dehydrogenases
3-Hydroxysteroid Dehydrogenases
Aldo-Keto Reductase Family 1 Member C3
Aldo-Keto Reductase Family 1 Member C3
Cell Line, Tumor
Cell Line, Tumor
Cell Proliferation
Cell Proliferation
Chromones
Chromones
Dinoprost
Dinoprost
Humans
Humans
Hydroxyprostaglandin Dehydrogenases
Hydroxyprostaglandin Dehydrogenases
Hydroxysteroid Dehydrogenases
Hydroxysteroid Dehydrogenases
Male
Male
Morpholines
Morpholines
Phosphatidylinositol 3-Kinases
Phosphatidylinositol 3-Kinases
Prostaglandin D2
Prostaglandin D2
Prostatic Neoplasms
Prostatic Neoplasms
Proto-Oncogene Proteins c-akt
Proto-Oncogene Proteins c-akt
Receptors, Prostaglandin
Receptors, Prostaglandin
Signal Transduction
Signal Transduction
Transfection
Transfection
authors with profiles
Kar-Ming Fung