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Exogenous CXCL4 infusion inhibits macrophage phagocytosis by limiting CD36 signalling to enhance post-myocardial infarction cardiac dilation and mortality.
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Exogenous CXCL4 infusion inhibits macrophage phagocytosis by limiting CD36 signalling to enhance post-myocardial infarction cardiac dilation and mortality.
Exogenous CXCL4 infusion inhibits macrophage phagocytosis by limiting CD36 signalling to enhance post-myocardial infarction cardiac dilation and mortality. Cardiovasc Res. 2019 02 01; 115(2):395-408.
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PubMed
subject areas
ADAMTS Proteins
ADAMTS Proteins
Animals
Animals
CD36 Antigens
CD36 Antigens
Cells, Cultured
Cells, Cultured
Disease Models, Animal
Disease Models, Animal
Humans
Humans
Hypertrophy, Left Ventricular
Hypertrophy, Left Ventricular
Infusions, Subcutaneous
Infusions, Subcutaneous
Integrin beta4
Integrin beta4
Macrophages, Peritoneal
Macrophages, Peritoneal
Male
Male
Matrix Metalloproteinase 9
Matrix Metalloproteinase 9
Mice, Inbred C57BL
Mice, Inbred C57BL
Myocardial Infarction
Myocardial Infarction
Myocardium
Myocardium
Phagocytosis
Phagocytosis
Platelet Factor 4
Platelet Factor 4
Signal Transduction
Signal Transduction
Ventricular Function, Left
Ventricular Function, Left
Ventricular Remodeling
Ventricular Remodeling
authors with profiles
Andriy Yabluchanskiy