Header Logo

Connection

Jimmy Ballard to Bacterial Proteins

Back to Details
This is a "connection" page, showing publications Jimmy Ballard has written about Bacterial Proteins.
Jimmy Ballard
Connection Strength
6.051
Bacterial Proteins
  1. A sequence invariable region in TcdB2 is required for toxin escape from Clostridioides difficile. J Bacteriol. 2024 07 25; 206(7):e0009624.
    View in: PubMed
    Score: 0.695
  2. Combined and Distinct Roles of Agr Proteins in Clostridioides difficile 630 Sporulation, Motility, and Toxin Production. mBio. 2020 12 22; 11(6).
    View in: PubMed
    Score: 0.546
  3. Deletion of a 19-Amino-Acid Region in Clostridioides difficile TcdB2 Results in Spontaneous Autoprocessing and Reduced Cell Binding and Provides a Nontoxic Immunogen for Vaccination. Infect Immun. 2019 08; 87(8).
    View in: PubMed
    Score: 0.495
  4. Cell-penetrating peptides derived from Clostridium difficile TcdB2 and a related large clostridial toxin. J Biol Chem. 2018 02 02; 293(5):1810-1819.
    View in: PubMed
    Score: 0.443
  5. Intrinsic Toxin-Derived Peptides Destabilize and Inactivate Clostridium difficile TcdB. mBio. 2017 05 16; 8(3).
    View in: PubMed
    Score: 0.425
  6. Exposure of neutralizing epitopes in the carboxyl-terminal domain of TcdB is altered by a proximal hypervariable region. J Biol Chem. 2015 Mar 13; 290(11):6975-85.
    View in: PubMed
    Score: 0.362
  7. Clostridium difficile 027/BI/NAP1 encodes a hypertoxic and antigenically variable form of TcdB. PLoS Pathog. 2013; 9(8):e1003523.
    View in: PubMed
    Score: 0.327
  8. TcdB from hypervirulent Clostridium difficile exhibits increased efficiency of autoprocessing. Mol Microbiol. 2012 Apr; 84(1):66-76.
    View in: PubMed
    Score: 0.296
  9. Variations in TcdB activity and the hypervirulence of emerging strains of Clostridium difficile. PLoS Pathog. 2010 Aug 19; 6(8):e1001061.
    View in: PubMed
    Score: 0.267
  10. Regulatory interactions of a virulence-associated serine/threonine phosphatase-kinase pair in Bacillus anthracis. J Bacteriol. 2010 Jan; 192(2):400-9.
    View in: PubMed
    Score: 0.253
  11. Clostridium difficile toxins: mechanism of action and role in disease. Clin Microbiol Rev. 2005 Apr; 18(2):247-63.
    View in: PubMed
    Score: 0.184
  12. Clostridioides difficile toxin B subverts germinal center and antibody recall responses by stimulating a drug-treatable CXCR4-dependent mechanism. Cell Rep. 2024 May 28; 43(5):114245.
    View in: PubMed
    Score: 0.173
  13. Mutational analysis of the enzymatic domain of Clostridium difficile toxin B reveals novel inhibitors of the wild-type toxin. Infect Immun. 2003 Jun; 71(6):3294-301.
    View in: PubMed
    Score: 0.162
  14. Clostridium difficile toxin B activates dual caspase-dependent and caspase-independent apoptosis in intoxicated cells. Cell Microbiol. 2002 Jul; 4(7):425-34.
    View in: PubMed
    Score: 0.152
  15. Identification of ClpP Dual Isoform Disruption as an Antisporulation Strategy for Clostridioides difficile. J Bacteriol. 2022 02 15; 204(2):e0041121.
    View in: PubMed
    Score: 0.145
  16. Autoinducing peptide-based quorum signaling systems in Clostridioides difficile. Curr Opin Microbiol. 2022 02; 65:81-86.
    View in: PubMed
    Score: 0.145
  17. Human C. difficile toxin-specific memory B cell repertoires encode poorly neutralizing antibodies. JCI Insight. 2020 08 20; 5(16).
    View in: PubMed
    Score: 0.133
  18. pH-induced conformational changes in Clostridium difficile toxin B. Infect Immun. 2000 May; 68(5):2470-4.
    View in: PubMed
    Score: 0.131
  19. Coordination between T helper cells, iNKT cells, and their follicular helper subsets in the humoral immune response against Clostridium difficile toxin B. J Leukoc Biol. 2017 02; 101(2):567-576.
    View in: PubMed
    Score: 0.101
  20. Memory B Cells Encode Neutralizing Antibody Specific for Toxin B from the Clostridium difficile Strains VPI 10463 and NAP1/BI/027 but with Superior Neutralization of VPI 10463 Toxin B. Infect Immun. 2016 01; 84(1):194-204.
    View in: PubMed
    Score: 0.095
  21. Variations in virulence and molecular biology among emerging strains of Clostridium difficile. Microbiol Mol Biol Rev. 2013 Dec; 77(4):567-81.
    View in: PubMed
    Score: 0.084
  22. The major neutralizing antibody responses to recombinant anthrax lethal and edema factors are directed to non-cross-reactive epitopes. Infect Immun. 2009 Nov; 77(11):4714-23.
    View in: PubMed
    Score: 0.062
  23. The mechanism of Bacillus anthracis intracellular germination requires multiple and highly diverse genetic loci. Infect Immun. 2009 Jan; 77(1):23-31.
    View in: PubMed
    Score: 0.059
  24. Functional comparison of the two Bacillus anthracis glutamate racemases. J Bacteriol. 2007 Jul; 189(14):5265-75.
    View in: PubMed
    Score: 0.053
  25. Elucidating the in vivo targets of bacterial toxins. Future Microbiol. 2007 Feb; 2(1):85-92.
    View in: PubMed
    Score: 0.052
  26. Genes that enhance the ecological fitness of Shewanella oneidensis MR-1 in sediments reveal the value of antibiotic resistance. Appl Environ Microbiol. 2007 Jan; 73(2):492-8.
    View in: PubMed
    Score: 0.051
  27. A method adapting microarray technology for signature-tagged mutagenesis of Desulfovibrio desulfuricans G20 and Shewanella oneidensis MR-1 in anaerobic sediment survival experiments. Appl Environ Microbiol. 2005 Nov; 71(11):7064-74.
    View in: PubMed
    Score: 0.048
  28. Response Regulator CD1688 Is a Negative Modulator of Sporulation in Clostridioides difficile. J Bacteriol. 2022 08 16; 204(8):e0013022.
    View in: PubMed
    Score: 0.038
  29. Clostridioides difficile Toxin B Activates Group 3 Innate Lymphocytes. Infect Immun. 2022 04 21; 90(4):e0007322.
    View in: PubMed
    Score: 0.037
  30. Desulfovibrio sp. genes involved in the respiration of sulfate during metabolism of hydrogen and lactate. Appl Environ Microbiol. 2002 Apr; 68(4):1932-7.
    View in: PubMed
    Score: 0.037
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.