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Connection

Jimmy Ballard to Cell Line

This is a "connection" page, showing publications Jimmy Ballard has written about Cell Line.
Connection Strength

1.007
  1. Unique, Intersecting, and Overlapping Roles of C/EBP ß and CREB in Cells of the Innate Immune System. Sci Rep. 2018 11 16; 8(1):16931.
    View in: PubMed
    Score: 0.137
  2. Increased cAMP in monocytes augments Notch signaling mechanisms by elevating RBP-J and transducin-like enhancer of Split (TLE). J Biol Chem. 2013 Jul 26; 288(30):21526-36.
    View in: PubMed
    Score: 0.094
  3. Glycogen synthase kinase 3 activation is important for anthrax edema toxin-induced dendritic cell maturation and anthrax toxin receptor 2 expression in macrophages. Infect Immun. 2011 Aug; 79(8):3302-8.
    View in: PubMed
    Score: 0.081
  4. Adenomatous polyposis coli protein associates with C/EBP beta and increases Bacillus anthracis edema toxin-stimulated gene expression in macrophages. J Biol Chem. 2011 Jun 03; 286(22):19364-72.
    View in: PubMed
    Score: 0.081
  5. Regulatory interactions of a virulence-associated serine/threonine phosphatase-kinase pair in Bacillus anthracis. J Bacteriol. 2010 Jan; 192(2):400-9.
    View in: PubMed
    Score: 0.073
  6. The mechanism of Bacillus anthracis intracellular germination requires multiple and highly diverse genetic loci. Infect Immun. 2009 Jan; 77(1):23-31.
    View in: PubMed
    Score: 0.068
  7. Critical intermediate steps in Clostridium sordellii lethal toxin-induced apoptosis. Biochem Biophys Res Commun. 2007 Nov 30; 363(4):959-64.
    View in: PubMed
    Score: 0.063
  8. Cytotoxic activity of Bacillus anthracis protective antigen observed in a macrophage cell line overexpressing ANTXR1. Cell Microbiol. 2006 Aug; 8(8):1272-81.
    View in: PubMed
    Score: 0.058
  9. Variations in lethal toxin and cholesterol-dependent cytolysin production correspond to differences in cytotoxicity among strains of Clostridium sordellii. FEMS Microbiol Lett. 2006 Jun; 259(2):295-302.
    View in: PubMed
    Score: 0.058
  10. Bacillus anthracis oedema toxin as a cause of tissue necrosis and cell type-specific cytotoxicity. Cell Microbiol. 2005 Aug; 7(8):1139-49.
    View in: PubMed
    Score: 0.055
  11. Toxin-induced resistance in Bacillus anthracis lethal toxin-treated macrophages. Proc Natl Acad Sci U S A. 2003 Oct 14; 100(21):12426-31.
    View in: PubMed
    Score: 0.048
  12. Decreased glycogen synthase kinase 3-beta levels and related physiological changes in Bacillus anthracis lethal toxin-treated macrophages. Cell Microbiol. 2003 Aug; 5(8):523-32.
    View in: PubMed
    Score: 0.047
  13. Clostridium difficile toxin B activates dual caspase-dependent and caspase-independent apoptosis in intoxicated cells. Cell Microbiol. 2002 Jul; 4(7):425-34.
    View in: PubMed
    Score: 0.044
  14. Memory B Cells Encode Neutralizing Antibody Specific for Toxin B from the Clostridium difficile Strains VPI 10463 and NAP1/BI/027 but with Superior Neutralization of VPI 10463 Toxin B. Infect Immun. 2016 01; 84(1):194-204.
    View in: PubMed
    Score: 0.028
  15. Select human anthrax protective antigen epitope-specific antibodies provide protection from lethal toxin challenge. J Infect Dis. 2010 Jul 15; 202(2):251-60.
    View in: PubMed
    Score: 0.019
  16. Resistance of human alveolar macrophages to Bacillus anthracis lethal toxin. J Immunol. 2009 Nov 01; 183(9):5799-806.
    View in: PubMed
    Score: 0.018
  17. Sequential B-cell epitopes of Bacillus anthracis lethal factor bind lethal toxin-neutralizing antibodies. Infect Immun. 2009 Jan; 77(1):162-9.
    View in: PubMed
    Score: 0.017
  18. High-throughput, single-cell analysis of macrophage interactions with fluorescently labeled Bacillus anthracis spores. Appl Environ Microbiol. 2008 Aug; 74(16):5201-10.
    View in: PubMed
    Score: 0.017
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.