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Connection

Sanjay Bidichandani to Trinucleotide Repeat Expansion

This is a "connection" page, showing publications Sanjay Bidichandani has written about Trinucleotide Repeat Expansion.
  1. Epigenetic promoter silencing in Friedreich ataxia is dependent on repeat length. Ann Neurol. 2014 Oct; 76(4):522-8.
    View in: PubMed
    Score: 0.455
  2. Pms2 suppresses large expansions of the (GAA·TTC)n sequence in neuronal tissues. PLoS One. 2012; 7(10):e47085.
    View in: PubMed
    Score: 0.399
  3. Repair of DNA double-strand breaks within the (GAA*TTC)n sequence results in frequent deletion of the triplet-repeat sequence. Nucleic Acids Res. 2008 Feb; 36(2):489-500.
    View in: PubMed
    Score: 0.285
  4. Anticipation and intergenerational repeat instability in spinocerebellar ataxia type 17. Ann Neurol. 2007 Jun; 61(6):607-10.
    View in: PubMed
    Score: 0.275
  5. Progressive GAA expansions in dorsal root ganglia of Friedreich's ataxia patients. Ann Neurol. 2007 Jan; 61(1):55-60.
    View in: PubMed
    Score: 0.268
  6. Replication in mammalian cells recapitulates the locus-specific differences in somatic instability of genomic GAA triplet-repeats. Nucleic Acids Res. 2006; 34(21):6352-61.
    View in: PubMed
    Score: 0.265
  7. The GAA triplet-repeat is unstable in the context of the human FXN locus and displays age-dependent expansions in cerebellum and DRG in a transgenic mouse model. Hum Genet. 2007 Jan; 120(5):633-40.
    View in: PubMed
    Score: 0.262
  8. Expansion of GAA trinucleotide repeats in mammals. Genomics. 2006 Jan; 87(1):57-67.
    View in: PubMed
    Score: 0.248
  9. Replication-mediated instability of the GAA triplet repeat mutation in Friedreich ataxia. Nucleic Acids Res. 2004; 32(19):5962-71.
    View in: PubMed
    Score: 0.231
  10. Genetic admixture of European FRDA genes is the cause of Friedreich ataxia in the Mexican population. Genomics. 2004 Nov; 84(5):779-84.
    View in: PubMed
    Score: 0.230
  11. Expansion of GAA triplet repeats in the human genome: unique origin of the FRDA mutation at the center of an Alu. Genomics. 2004 Mar; 83(3):373-83.
    View in: PubMed
    Score: 0.220
  12. DNA methylation in Friedreich ataxia silences expression of frataxin isoform E. Sci Rep. 2022 03 23; 12(1):5031.
    View in: PubMed
    Score: 0.192
  13. Very late-onset Friedreich ataxia despite large GAA triplet repeat expansions. Arch Neurol. 2000 Feb; 57(2):246-51.
    View in: PubMed
    Score: 0.166
  14. Somatic sequence variation at the Friedreich ataxia locus includes complete contraction of the expanded GAA triplet repeat, significant length variation in serially passaged lymphoblasts and enhanced mutagenesis in the flanking sequence. Hum Mol Genet. 1999 Dec; 8(13):2425-36.
    View in: PubMed
    Score: 0.164
  15. Reversal of epigenetic promoter silencing in Friedreich ataxia by a class I histone deacetylase inhibitor. Nucleic Acids Res. 2016 06 20; 44(11):5095-104.
    View in: PubMed
    Score: 0.126
  16. Role of transcript and interplay between transcription and replication in triplet-repeat instability in mammalian cells. Nucleic Acids Res. 2011 Jan; 39(2):526-35.
    View in: PubMed
    Score: 0.086
  17. Length-dependent structure formation in Friedreich ataxia (GAA)n*(TTC)n repeats at neutral pH. Nucleic Acids Res. 2004; 32(3):1224-31.
    View in: PubMed
    Score: 0.055
  18. The GAA triplet-repeat sequence in Friedreich ataxia shows a high level of somatic instability in vivo, with a significant predilection for large contractions. Hum Mol Genet. 2002 Sep 01; 11(18):2175-87.
    View in: PubMed
    Score: 0.050
  19. Distinct distribution of autosomal dominant spinocerebellar ataxia in the Mexican population. Mov Disord. 2007 May 15; 22(7):1050-3.
    View in: PubMed
    Score: 0.017
Connection Strength

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Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.