Header Logo

Connection

Co-Authors

This is a "connection" page, showing publications co-authored by Trent Tipple and Lynette Rogers.
Connection Strength

5.087
  1. Attenuation of miR-17~92 Cluster in Bronchopulmonary Dysplasia. Ann Am Thorac Soc. 2015 Oct; 12(10):1506-13.
    View in: PubMed
    Score: 0.523
  2. Methods for the determination of plasma or tissue glutathione levels. Methods Mol Biol. 2012; 889:315-24.
    View in: PubMed
    Score: 0.404
  3. Maternal docosahexaenoic acid supplementation decreases lung inflammation in hyperoxia-exposed newborn mice. J Nutr. 2011 Feb; 141(2):214-22.
    View in: PubMed
    Score: 0.376
  4. Alterations of the thioredoxin system by hyperoxia: implications for alveolar development. Am J Respir Cell Mol Biol. 2009 Nov; 41(5):612-9.
    View in: PubMed
    Score: 0.331
  5. Selenium Deficiency Exacerbates Hyperoxia-Induced Lung Injury in Newborn C3H/HeN Mice. Antioxidants (Basel). 2024 Mar 25; 13(4).
    View in: PubMed
    Score: 0.236
  6. Effects of DNA methylase inhibitors in a murine model of severe BPD. Respir Physiol Neurobiol. 2023 Apr 07; 313:104060.
    View in: PubMed
    Score: 0.220
  7. Transcriptomic-Metabolomic Profiling in Mouse Lung Tissues Reveals Sex- and Strain-Based Differences. Metabolites. 2022 Sep 30; 12(10).
    View in: PubMed
    Score: 0.213
  8. Auranofin-Mediated NRF2 Induction Attenuates Interleukin 1 Beta Expression in Alveolar Macrophages. Antioxidants (Basel). 2021 Apr 21; 10(5).
    View in: PubMed
    Score: 0.192
  9. Glutathione reductase deficiency alters lung development and hyperoxic responses in neonatal mice. Redox Biol. 2021 01; 38:101797.
    View in: PubMed
    Score: 0.187
  10. Thioredoxin Reductase-1 Inhibition Augments Endogenous Glutathione-Dependent Antioxidant Responses in Experimental Bronchopulmonary Dysplasia. Oxid Med Cell Longev. 2019; 2019:7945983.
    View in: PubMed
    Score: 0.165
  11. Alterations in VASP phosphorylation and profilin1 and cofilin1 expression in hyperoxic lung injury and BPD. Respir Res. 2018 Nov 21; 19(1):229.
    View in: PubMed
    Score: 0.163
  12. Aurothioglucose does not improve alveolarization or elicit sustained Nrf2 activation in C57BL/6 models of bronchopulmonary dysplasia. Am J Physiol Lung Cell Mol Physiol. 2018 05 01; 314(5):L736-L742.
    View in: PubMed
    Score: 0.154
  13. miR-29b supplementation decreases expression of matrix proteins and improves alveolarization in mice exposed to maternal inflammation and neonatal hyperoxia. Am J Physiol Lung Cell Mol Physiol. 2017 08 01; 313(2):L339-L349.
    View in: PubMed
    Score: 0.146
  14. Of mice and men: correlations between microRNA-17~92 cluster expression and promoter methylation in severe bronchopulmonary dysplasia. Am J Physiol Lung Cell Mol Physiol. 2016 Nov 01; 311(5):L981-L984.
    View in: PubMed
    Score: 0.140
  15. Thioredoxin Reductase Inhibition Attenuates Neonatal Hyperoxic Lung Injury and Enhances Nuclear Factor E2-Related Factor 2 Activation. Am J Respir Cell Mol Biol. 2016 09; 55(3):419-28.
    View in: PubMed
    Score: 0.139
  16. Maternal high fat diet exposure is associated with increased hepcidin levels, decreased myelination, and neurobehavioral changes in male offspring. Brain Behav Immun. 2016 Nov; 58:369-378.
    View in: PubMed
    Score: 0.139
  17. DHA suppresses chronic apoptosis in the lung caused by perinatal inflammation. Am J Physiol Lung Cell Mol Physiol. 2015 Sep 01; 309(5):L441-8.
    View in: PubMed
    Score: 0.129
  18. The thioredoxin reductase-1 inhibitor aurothioglucose attenuates lung injury and improves survival in a murine model of acute respiratory distress syndrome. Antioxid Redox Signal. 2014 Jun 10; 20(17):2681-91.
    View in: PubMed
    Score: 0.117
  19. Maternal dietary docosahexaenoic acid supplementation attenuates fetal growth restriction and enhances pulmonary function in a newborn mouse model of perinatal inflammation. J Nutr. 2014 Mar; 144(3):258-66.
    View in: PubMed
    Score: 0.116
  20. Neonatal hyperoxic exposure persistently alters lung secretoglobins and annexin A1. Biomed Res Int. 2013; 2013:408485.
    View in: PubMed
    Score: 0.114
  21. Cyclooxygenase-2 in newborn hyperoxic lung injury. Free Radic Biol Med. 2013 Aug; 61:502-11.
    View in: PubMed
    Score: 0.111
  22. Thioredoxin reductase inhibition elicits Nrf2-mediated responses in Clara cells: implications for oxidant-induced lung injury. Antioxid Redox Signal. 2012 Nov 15; 17(10):1407-16.
    View in: PubMed
    Score: 0.104
  23. Prenatal inflammation exacerbates hyperoxia-induced functional and structural changes in adult mice. Am J Physiol Regul Integr Comp Physiol. 2012 Aug 01; 303(3):R279-90.
    View in: PubMed
    Score: 0.104
  24. Plasma lipid metabolites are associated with gestational age but not bronchopulmonary dysplasia. Acta Paediatr. 2012 Aug; 101(8):e321-6.
    View in: PubMed
    Score: 0.103
  25. Lipopolysaccharide-induced cyclooxygenase-2 expression in mouse transformed Clara cells. Cell Physiol Biochem. 2012; 29(1-2):213-22.
    View in: PubMed
    Score: 0.102
  26. Thioredoxin-interacting protein inhibits hypoxia-inducible factor transcriptional activity. Free Radic Biol Med. 2010 Nov 15; 49(9):1361-7.
    View in: PubMed
    Score: 0.092
  27. Hyperoxia exposure alters hepatic eicosanoid metabolism in newborn mice. Pediatr Res. 2010 Feb; 67(2):144-9.
    View in: PubMed
    Score: 0.088
  28. Differential responses in the lungs of newborn mouse pups exposed to 85% or >95% oxygen. Pediatr Res. 2009 Jan; 65(1):33-8.
    View in: PubMed
    Score: 0.082
  29. Thioredoxin-related mechanisms in hyperoxic lung injury in mice. Am J Respir Cell Mol Biol. 2007 Oct; 37(4):405-13.
    View in: PubMed
    Score: 0.074
  30. Riboflavin supplementation does not attenuate hyperoxic lung injury in transgenic (spc-mt)hGR mice. Exp Lung Res. 2011 Apr; 37(3):155-61.
    View in: PubMed
    Score: 0.023
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.