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Novel therapeutic approaches to lupus glomerulonephritis: translating animal models to clinical practice.
Deficiency of a transcriptional regulator, inhibitor of differentiation 3, induces glomerulonephritis in apolipoprotein E-deficient mice: a model linking hyperlipidemia and renal disease.
Inhibitor of differentiation 3, a transcription factor, regulates hyperlipidemia-associated kidney disease.
Influenza A (H1N1) virus infection triggers severe pulmonary inflammation in lupus-prone mice following viral clearance.
Activated CD4+ T cells target mesangial antigens and initiate glomerulonephritis.
Anti-alpha8 integrin immunoliposomes in glomeruli of lupus-susceptible mice: a novel system for delivery of therapeutic agents to the renal glomerulus in systemic lupus erythematosus.
Pathogenesis of kidney disease in systemic lupus erythematosus.
Mesangial pathology in glomerular disease: targets for therapeutic intervention.
Cgnz1 allele confers kidney resistance to damage preventing progression of immune complex-mediated acute lupus glomerulonephritis.
Loss of mucin-type O-glycans impairs the integrity of the glomerular filtration barrier in the mouse kidney.
Failure of CD25+ T cells from lupus-prone mice to suppress lupus glomerulonephritis and sialoadenitis.