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One or more keywords matched the following items that are connected to Zhao, Zhizhuang
Item TypeName
Concept Mutation
Concept Point Mutation
Concept Mutation, Missense
Academic Article Transgenic expression of JAK2V617F causes myeloproliferative disorders in mice.
Academic Article Generation and characterization of a highly effective protein substrate for analysis of FLT3 activity.
Academic Article JAK2V617F and p53 mutations coexist in erythroleukemia and megakaryoblastic leukemic cell lines.
Academic Article Thrombopoietin is required for full phenotype expression in a JAK2V617F transgenic mouse model of polycythemia vera.
Academic Article Role of tyrosine kinases and phosphatases in polycythemia vera.
Academic Article Characterization of a highly effective protein substrate for analysis of JAK2(V617F) Activity.
Academic Article Development of a highly sensitive method for detection of JAK2V617F.
Academic Article RBPJ mutations identified in two families affected by Adams-Oliver syndrome.
Academic Article Cardiac hypertrophy associated with myeloproliferative neoplasms in JAK2V617F transgenic mice.
Academic Article Generation and characterization of a JAK2V617F-containing erythroleukemia cell line.
Academic Article Blocking the function of tyrosine phosphatase SHP-2 by targeting its Src homology 2 domains.
Academic Article Identification of an acquired JAK2 mutation in polycythemia vera.
Academic Article JAK2(V617F): Prevalence in a large Chinese hospital population.
Academic Article Detection of the JAK2V617F mutation by asymmetric PCR and melt curve analysis.
Academic Article Myotubularin family phosphatase ceMTM3 is required for muscle maintenance by preventing excessive autophagy in Caenorhabditis elegans.
Academic Article ASXL1 alteration cooperates with JAK2V617F to accelerate myelofibrosis.
Academic Article miR-543 regulates the epigenetic landscape of myelofibrosis by targeting TET1 and TET2.
Grant Tyrosine Phosphatase PTP-MEG2 and Hematopoiesis
Grant Tyrosine Kinase JAK2 and Myeloproliferative Disorders
Academic Article Leukemogenic SHP2 mutations lead to erythropoietin independency of HCD-57 cells: a novel model for preclinical research of SHP2-mutant JMML.
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