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Bacillus anthracis lethal toxin reduces human alveolar epithelial barrier function.
Resistance of human alveolar macrophages to Bacillus anthracis lethal toxin.
Bacillus anthracis spore movement does not require a carrier cell and is not affected by lethal toxin in human lung models.
Anthrax Edema and Lethal Toxins Differentially Target Human Lung and Blood Phagocytes.
Bacillus anthracis spores stimulate cytokine and chemokine innate immune responses in human alveolar macrophages through multiple mitogen-activated protein kinase pathways.
Human lung innate immune response to Bacillus anthracis spore infection.
Gene expression profiling of human alveolar macrophages infected by B. anthracis spores demonstrates TNF-alpha and NF-kappab are key components of the innate immune response to the pathogen.
Gene expression profiling of primary human type I alveolar epithelial cells exposed to Bacillus anthracis spores reveals induction of neutrophil and monocyte chemokines.