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Influenza A Virus, H1N1 Subtype
Innate immune response to H3N2 and H1N1 influenza virus infection in a human lung organ culture model.
Influenza A(H1N1)pdm09 virus suppresses RIG-I initiated innate antiviral responses in the human lung.
Human primary airway epithelial cells isolated from active smokers have epigenetically impaired antiviral responses.
RIG-I and TLR3 are both required for maximum interferon induction by influenza virus in human lung alveolar epithelial cells.
Cigarette smoke extract suppresses the RIG-I-initiated innate immune response to influenza virus in the human lung.
Cigarette smoke attenuates the RIG-I-initiated innate antiviral response to influenza infection in two murine models.
Long-term cigarette smoke exposure dysregulates pulmonary T cell response and IFN-? protection to influenza virus in mouse.
Early IFN-ß administration protects cigarette smoke exposed mice against lethal influenza virus infection without increasing lung inflammation.
Influenza A Virus H1N1 Subtype