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Interferon Type I
Interferon Regulatory Factor-7
Influenza A(H1N1)pdm09 virus suppresses RIG-I initiated innate antiviral responses in the human lung.
Human primary airway epithelial cells isolated from active smokers have epigenetically impaired antiviral responses.
RIG-I and TLR3 are both required for maximum interferon induction by influenza virus in human lung alveolar epithelial cells.
The Role of Type I IFNs in Influenza: Antiviral Superheroes or Immunopathogenic Villains?
IRF7 Is Required for the Second Phase Interferon Induction during Influenza Virus Infection in Human Lung Epithelia.
Cigarette smoke extract suppresses the RIG-I-initiated innate immune response to influenza virus in the human lung.
Cigarette smoke attenuates the RIG-I-initiated innate antiviral response to influenza infection in two murine models.
Reversing Cigarette Smoke-Induced Antiviral Immunosuppression
Long-term cigarette smoke exposure dysregulates pulmonary T cell response and IFN-? protection to influenza virus in mouse.
Early IFN-ß administration protects cigarette smoke exposed mice against lethal influenza virus infection without increasing lung inflammation.