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Memory B Cells Encode Neutralizing Antibody Specific for Toxin B from the Clostridium difficile Strains VPI 10463 and NAP1/BI/027 but with Superior Neutralization of VPI 10463 Toxin B.
Decreased SMG7 expression associates with lupus-risk variants and elevated antinuclear antibody production.
Protective antigen-specific memory B cells persist years after anthrax vaccination and correlate with humoral immunity.
Deep sequencing reveals a DAP1 regulatory haplotype that potentiates autoimmunity in systemic lupus erythematosus.