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STAT1 signaling within macrophages is required for antifungal activity against Cryptococcus neoformans.
IFN-? immune priming of macrophages in vivo induces prolonged STAT1 binding and protection against Cryptococcus neoformans.
Pulmonary infection with an interferon-gamma-producing Cryptococcus neoformans strain results in classical macrophage activation and protection.
Pulmonary Macrophage and Dendritic Cell Responses to Cryptococcus neoformans.
In vivo role of dendritic cells in a murine model of pulmonary cryptococcosis.
Protective immunity against pulmonary cryptococcosis is associated with STAT1-mediated classical macrophage activation.
Interleukin-17 is not required for classical macrophage activation in a pulmonary mouse model of Cryptococcus neoformans infection.
STAT1 signaling is essential for protection against Cryptococcus neoformans infection in mice.
Transcriptional Changes in Pulmonary Phagocyte Subsets Dictate the Outcome Following Interaction With The Fungal Pathogen Cryptococcus neoformans.