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A TMEFF2-regulated cell cycle derived gene signature is prognostic of recurrence risk in prostate cancer.
5alpha-androstane-3alpha,17beta-diol supports human prostate cancer cell survival and proliferation through androgen receptor-independent signaling pathways: implication of androgen-independent prostate cancer progression.
Androgen receptor signaling is required for androgen-sensitive human prostate cancer cell proliferation and survival.
Increased expression of type 2 3alpha-hydroxysteroid dehydrogenase/type 5 17beta-hydroxysteroid dehydrogenase (AKR1C3) and its relationship with androgen receptor in prostate carcinoma.
Elevated AKR1C3 expression promotes prostate cancer cell survival and prostate cell-mediated endothelial cell tube formation: implications for prostate cancer progression.
Analysis of TMEFF2 allografts and transgenic mouse models reveals roles in prostate regeneration and cancer.
AKR1C2 and AKR1C3 mediated prostaglandin D2 metabolism augments the PI3K/Akt proliferative signaling pathway in human prostate cancer cells.
Linking ?-aminobutyric acid A receptor to epidermal growth factor receptor pathways activation in human prostate cancer.
Transition from androgenic to neurosteroidal action of 5a-androstane-3a, 17ß-diol through the type A ?-aminobutyric acid receptor in prostate cancer progression.
Characterization of a monoclonal antibody for human aldo-keto reductase AKR1C3 (type 2 3alpha-hydroxysteroid dehydrogenase/type 5 17beta-hydroxysteroid dehydrogenase); immunohistochemical detection in breast and prostate.
Aldo-keto reductase (AKR) 1C3: role in prostate disease and the development of specific inhibitors.