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Cyclooxygenase 2 Inhibitors
Intestinal tuft cells regulate the ATM mediated DNA Damage response via Dclk1 dependent mechanism for crypt restitution following radiation injury.
Disruption of cyclooxygenase-1 gene results in an impaired response to radiation injury.
Coupled mRNA stabilization and translational silencing of cyclooxygenase-2 by a novel RNA binding protein, CUGBP2.
Cyclooxygenase expression in intestinal epithelial cells.
Azoxymethane protects intestinal stem cells and reduces crypt epithelial mitosis through a COX-1-dependent mechanism.
Novel intestinal splice variants of RNA-binding protein CUGBP2: isoform-specific effects on mitotic catastrophe.
Diphenyl difluoroketone: a curcumin derivative with potent in vivo anticancer activity.
HuR and TTP: two RNA binding proteins that deliver message from the 3' end.
Dynamic antagonism between RNA-binding protein CUGBP2 and cyclooxygenase-2-mediated prostaglandin E2 in radiation damage.
Gastrin-mediated interleukin-8 and cyclooxygenase-2 gene expression: differential transcriptional and posttranscriptional mechanisms.
Translation regulatory factor RBM3 is a proto-oncogene that prevents mitotic catastrophe.
Targeting pancreatitis blocks tumor-initiating stem cells and pancreatic cancer progression.
CUGBP2 plays a critical role in apoptosis of breast cancer cells in response to genotoxic injury.
Apobec-1 protects intestine from radiation injury through posttranscriptional regulation of cyclooxygenase-2 expression.
Functional antagonism between RNA binding proteins HuR and CUGBP2 determines the fate of COX-2 mRNA translation.
Clinical implications of prostaglandin inhibition in the small bowel.
RNA binding protein CUGBP2/CELF2 mediates curcumin-induced mitotic catastrophe of pancreatic cancer cells.
Prostaglandin Endoperoxide Synthases