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Posthemorrhagic shock mesenteric lymph primes circulating neutrophils and provokes lung injury.
Blood transfusion and the two-insult model of post-injury multiple organ failure.
Hypertonic saline activation of p38 MAPK primes the PMN respiratory burst.
Hypertonic saline inhibits neutrophil (PMN) priming via attenuation of p38 MAPK signaling.
The lipid fraction of post-hemorrhagic shock mesenteric lymph (PHSML) inhibits neutrophil apoptosis and enhances cytotoxic potential.