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Rats, Inbred F344
Rats, Inbred WKY
Cognitive differences between Sprague-Dawley rats selectively bred for sensitivity or resistance to diet induced obesity.
Stereotaxic delivery of corticosterone to the amygdala modulates colonic sensitivity in rats.
Stereotaxic localization of corticosterone to the amygdala enhances hypothalamo-pituitary-adrenal responses to behavioral stress.
Corticosterone acts directly at the amygdala to alter spinal neuronal activity in response to colorectal distension.
Gestational hypoxia disrupts the neonatal leptin surge and programs hyperphagia and obesity in male offspring in the Sprague-Dawley rat.
Corticosterone delivery to the amygdala increases corticotropin-releasing factor mRNA in the central amygdaloid nucleus and anxiety-like behavior.
Corticotropin-releasing factor 1 receptor-mediated mechanisms inhibit colonic hypersensitivity in rats.
Strain differences in anxiety-like behavior: association with corticotropin-releasing factor.
Evidence for visceral hypersensitivity in high-anxiety rats.
Corticosterone implants to the amygdala and type 1 CRH receptor regulation: effects on behavior and colonic sensitivity.
Long-term expression of corticotropin-releasing factor (CRF) in the paraventricular nucleus of the hypothalamus in response to an acute colonic inflammation.
Chronically elevated corticosterone in the amygdala increases corticotropin releasing factor mRNA in the dorsolateral bed nucleus of stria terminalis following duress.
In utero programming of CRF neurons by glucocorticoids