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One or more keywords matched the following items that are connected to Xia, Lijun
Item TypeName
Concept Platelet Glycoprotein GPIIb-IIIa Complex
Concept Spike Glycoprotein, Coronavirus
Concept Membrane Glycoproteins
Academic Article P-selectin glycoprotein ligand-1-deficient mice have impaired leukocyte tethering to E-selectin under flow.
Academic Article Core 1-derived O-glycans are essential E-selectin ligands on neutrophils.
Academic Article Podoplanin maintains high endothelial venule integrity by interacting with platelet CLEC-2.
Academic Article Podoplanin requires sialylated O-glycans for stable expression on lymphatic endothelial cells and for interaction with platelets.
Academic Article L-selectin mechanochemistry restricts neutrophil priming in vivo.
Academic Article Cytoplasmic domain of P-selectin glycoprotein ligand-1 facilitates dimerization and export from the endoplasmic reticulum.
Academic Article P-selectin glycoprotein ligand-1 is highly expressed on Ly-6Chi monocytes and a major determinant for Ly-6Chi monocyte recruitment to sites of atherosclerosis in mice.
Academic Article Separable requirements for cytoplasmic domain of PSGL-1 in leukocyte rolling and signaling under flow.
Academic Article O-glycans direct selectin ligands to lipid rafts on leukocytes.
Academic Article N-terminal residues in murine P-selectin glycoprotein ligand-1 required for binding to murine P-selectin.
Academic Article Carcinoma mucins trigger reciprocal activation of platelets and neutrophils in a murine model of Trousseau syndrome.
Academic Article CLEC-2 and podoplanin, partners again.
Grant Protein-glycan interactions in the vascular system
Grant Podoplanin-mediated platelet activation and vascular integrity in the developing brain
Grant Platelet CLEC-2 in Arterial Thrombosis
Academic Article Heightened activation of embryonic megakaryocytes causes aneurysms in the developing brain of mice lacking podoplanin.
Academic Article L-SIGN is a receptor on liver sinusoidal endothelial cells for SARS-CoV-2 virus.
Academic Article Deletion of platelet CLEC-2 decreases GPIba-mediated integrin aIIbß3 activation and decreases thrombosis in TTP.
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