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Interferon Type I
Receptor, Interferon alpha-beta
Interferon Regulatory Factors
Estrogen receptor signaling promotes dendritic cell differentiation by increasing expression of the transcription factor IRF4.
IRF4-dependent dendritic cells regulate CD8+ T-cell differentiation and memory responses in influenza infection.
Deficiency of type I IFN receptor in lupus-prone New Zealand mixed 2328 mice decreases dendritic cell numbers and activation and protects from disease.
IRF4 and IRF8 Act in CD11c+ Cells To Regulate Terminal Differentiation of Lung Tissue Dendritic Cells.
Augmenting E Protein Activity Impairs cDC2 Differentiation at the Pre-cDC Stage.
IRF4 promotes cutaneous dendritic cell migration to lymph nodes during homeostasis and inflammation.
Increased level of E protein activity during invariant NKT development promotes differentiation of invariant NKT2 and invariant NKT17 subsets.
Identification of a Sjögren's syndrome susceptibility locus at OAS1 that influences isoform switching, protein expression, and responsiveness to type I interferons.
Presentation of arthritogenic peptide to antigen-specific T cells by fibroblast-like synoviocytes.
Estrogen receptors regulate innate immune cells and signaling pathways.
Sex Differences and Estrogen Receptors Regulate Dendritic Cells in Inflammation
IRF4-Mediated Regulation of Lung Dendritic Cells During Viral Infection
Long-term cigarette smoke exposure dysregulates pulmonary T cell response and IFN-? protection to influenza virus in mouse.