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West Nile virus-infected human dendritic cells fail to fully activate invariant natural killer T cells.
IRF4-dependent dendritic cells regulate CD8+ T-cell differentiation and memory responses in influenza infection.
Estrogen preferentially promotes the differentiation of CD11c+ CD11b(intermediate) dendritic cells from bone marrow precursors.
Increased level of E protein activity during invariant NKT development promotes differentiation of invariant NKT2 and invariant NKT17 subsets.
The immune response to herpes simplex virus type 1 infection in susceptible mice is a major cause of central nervous system pathology resulting in fatal encephalitis.