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Interferon Type I
Receptor, Interferon alpha-beta
Salivary gland hypofunction induced by activation of innate immunity is dependent on type I interferon signaling.
Type I interferon receptor deficiency prevents murine Sjogren's syndrome.
Activation of innate immune responses through Toll-like receptor 3 causes a rapid loss of salivary gland function.
Genetic complementation results in augmented autoantibody responses to lupus-associated antigens.
Activation of Stimulator of Interferon Genes (STING) and Sjögren Syndrome.
Pulmonary Involvement in a Mouse Model of Sjögren's Syndrome Induced by STING Activation.
Protocols for Experimental Sjögren's Syndrome.
Natural IgM anti-leukocyte autoantibodies attenuate excess inflammation mediated by innate and adaptive immune mechanisms involving Th-17.
Innate Immunity Activation In Pathogenesis of Sjogren's Syndrome
Cytosolic DNA sensing pathway in the pathogenesis of Sjogren's Syndrome