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Nicotinamide mononucleotide (NMN) supplementation promotes anti-aging miRNA expression profile in the aorta of aged mice, predicting epigenetic rejuvenation and anti-atherogenic effects.
Genome-wide DNA methylation study suggests epigenetic accessibility and transcriptional poising of interferon-regulated genes in naïve CD4+ T cells from lupus patients.
Epigenetic Reprogramming in Naive CD4+ T Cells Favoring T Cell Activation and Non-Th1 Effector T Cell Immune Response as an Early Event in Lupus Flares.
CD4+CD28+KIR+CD11ahi T cells correlate with disease activity and are characterized by a pro-inflammatory epigenetic and transcriptional profile in lupus patients.
The role of DNA methylation in epigenetics of aging.
Epigenomic elements enriched in the promoters of autoimmunity susceptibility genes.
Systematic classification of non-coding RNAs by epigenomic similarity.
Absence of genomic hypomethylation or regulation of cytosine-modifying enzymes with aging in male and female mice.
Data-mining analysis suggests an epigenetic pathogenesis for type 2 diabetes.
Genome-wide DNA methylation patterns in CD4+ T cells from patients with systemic lupus erythematosus.
Epigenome profiling reveals significant DNA demethylation of interferon signature genes in lupus neutrophils.
Ethnicity-specific epigenetic variation in naïve CD4+ T cells and the susceptibility to autoimmunity.
Oklahoma Nathan Shock Center of Excellence in Basic Biology of Aging
Many chronological aging clocks can be found throughout the epigenome: Implications for quantifying biological aging.
Multi-tissue DNA methylation microarray signature is predictive of gene function.
Hypomethylation of miR-17-92 cluster in lupus T cells and no significant role for genetic factors in the lupus-associated DNA methylation signature.