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Bacterial Outer Membrane Proteins
PhoPQ regulates acidic glycerophospholipid content of the Salmonella Typhimurium outer membrane.
Salmonellae PhoPQ regulation of the outer membrane to resist innate immunity.
Salmonella enterica Serovar Typhimurium Uses PbgA/YejM To Regulate Lipopolysaccharide Assembly during Bacteremia.
Delivery of cardiolipins to the Salmonella outer membrane is necessary for survival within host tissues and virulence.
Outer Membrane Lipid Secretion and the Innate Immune Response to Gram-Negative Bacteria.
Cues from the Membrane: Bacterial Glycerophospholipids.
Salmonella Tol-Pal Reduces Outer Membrane Glycerophospholipid Levels for Envelope Homeostasis and Survival during Bacteremia.
The Acinetobacter baumannii Mla system and glycerophospholipid transport to the outer membrane.
Separation of the Cell Envelope for Gram-negative Bacteria into Inner and Outer Membrane Fractions with Technical Adjustments for Acinetobacter baumannii.
Mechanisms of PbgA involvement in Salmonella Typhimurium lipid homeostasis for bacterial pathogenesis
The role of PhoP/PhoQ-regulated phospholipid alterations in Salmonella pathogenes
Conserved Tandem Arginines for PbgA/YejM Allow Salmonella Typhimurium To Regulate LpxC and Control Lipopolysaccharide Biogenesis during Infection.