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Enterococcal surface protein Esp does not facilitate intestinal colonization or translocation of Enterococcus faecalis in clindamycin-treated mice.
The N-terminal domain of enterococcal surface protein, Esp, is sufficient for Esp-mediated biofilm enhancement in Enterococcus faecalis.
Putative surface proteins encoded within a novel transferable locus confer a high-biofilm phenotype to Enterococcus faecalis.
Role of Enterococcus faecalis surface protein Esp in the pathogenesis of ascending urinary tract infection.
A novel putative enterococcal pathogenicity island linked to the esp virulence gene of Enterococcus faecium and associated with epidemicity.
Enterococcal surface protein, Esp, enhances biofilm formation by Enterococcus faecalis.
Surface protein Esp enhances pro-inflammatory cytokine expression through NF-?B activation during enterococcal infection.