Item Type | Name |
Concept
|
B-Lymphocytes
|
Concept
|
Dendritic Cells
|
Concept
|
Hematopoietic Stem Cells
|
Concept
|
Lymphocytes
|
Concept
|
Plasma Cells
|
Concept
|
T-Lymphocytes
|
Concept
|
Receptors, Antigen, B-Cell
|
Concept
|
Stem Cells
|
Concept
|
Gene Rearrangement, B-Lymphocyte, Heavy Chain
|
Concept
|
CHO Cells
|
Concept
|
K562 Cells
|
Concept
|
Tumor Cells, Cultured
|
Concept
|
B-Lymphocyte Subsets
|
Concept
|
Pluripotent Stem Cells
|
Concept
|
Adult Stem Cells
|
Concept
|
Precursor Cells, B-Lymphoid
|
Concept
|
Cells, Cultured
|
Academic Article
|
Serum amyloid A protects murine macrophages from lethal toxin-mediated death.
|
Academic Article
|
Insights From Analysis of Human Antigen-Specific Memory B Cell Repertoires.
|
Academic Article
|
The transcription factor, Bright, is not expressed in all human B lymphocyte subpopulations.
|
Academic Article
|
The transcription factor Bright plays a role in marginal zone B lymphocyte development and autoantibody production.
|
Academic Article
|
ARID3a gene profiles are strongly associated with human interferon alpha production.
|
Academic Article
|
ARID3a expression in human hematopoietic stem cells is associated with distinct gene patterns in aged individuals.
|
Academic Article
|
Bruton's tyrosine kinase regulates immunoglobulin promoter activation in association with the transcription factor Bright.
|
Academic Article
|
Transgenic mice expressing dominant-negative bright exhibit defects in B1 B cells.
|
Academic Article
|
The ARID family transcription factor bright is required for both hematopoietic stem cell and B lineage development.
|
Academic Article
|
Antibody Reactivity of B Cells in Lupus Patients with Increased Disease Activity and ARID3a Expression.
|
Academic Article
|
TLR engagement induces ARID3a in human blood hematopoietic progenitors and modulates IFNa production.
|
Academic Article
|
High frequency of matrix attachment regions and cut-like protein x/CCAAT-displacement protein and B cell regulator of IgH transcription binding sites flanking Ig V region genes.
|
Academic Article
|
Mutations in the DNA-binding domain of the transcription factor Bright act as dominant negative proteins and interfere with immunoglobulin transactivation.
|
Academic Article
|
Induction of immunoglobulin heavy-chain transcription through the transcription factor Bright requires TFII-I.
|
Academic Article
|
Anti-nuclear antibody production and autoimmunity in transgenic mice that overexpress the transcription factor Bright.
|
Academic Article
|
Loss of Bright/ARID3a function promotes developmental plasticity.
|
Academic Article
|
Differential expression of the transcription factor ARID3a in lupus patient hematopoietic progenitor cells.
|
Academic Article
|
A developmentally plastic adult mouse kidney cell line spontaneously generates multiple adult kidney structures.
|
Academic Article
|
Retinoids accelerate B lineage lymphoid differentiation.
|
Academic Article
|
Signalling of the BCR is regulated by a lipid rafts-localised transcription factor, Bright.
|
Academic Article
|
Disease activity in systemic lupus erythematosus correlates with expression of the transcription factor AT-rich-interactive domain 3A.
|
Academic Article
|
The Transcription Factor ARID3a Is Important for In Vitro Differentiation of Human Hematopoietic Progenitors.
|
Academic Article
|
Human effector B lymphocytes express ARID3a and secrete interferon alpha.
|
Academic Article
|
New Frontiers: ARID3a in SLE.
|
Grant
|
Role of the transcription factor ARID3a in innate immune responses
|
Grant
|
Role of the transcription factor ARID3a in lupus
|
Grant
|
Identification of Proteins Interacting with ARID3a
|
Grant
|
Role of the Transcription Factor ARID3a in Innate Immune Responses
|
Grant
|
Bright Function in the Immune System
|
Academic Article
|
Deficiencies in the DNA Binding Protein ARID3a Alter Chromatin Structures Important for Early Human Erythropoiesis.
|