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The transcription factor Bright plays a role in marginal zone B lymphocyte development and autoantibody production.
Transgenic mice expressing dominant-negative bright exhibit defects in B1 B cells.
TLR engagement induces ARID3a in human blood hematopoietic progenitors and modulates IFNa production.
Disease activity in systemic lupus erythematosus correlates with expression of the transcription factor AT-rich-interactive domain 3A.
The Transcription Factor ARID3a Is Important for In Vitro Differentiation of Human Hematopoietic Progenitors.
Human effector B lymphocytes express ARID3a and secrete interferon alpha.