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TOR Serine-Threonine Kinases
mTOR drives cerebrovascular, synaptic, and cognitive dysfunction in normative aging.
Over-expression of heat shock factor 1 phenocopies the effect of chronic inhibition of TOR by rapamycin and is sufficient to ameliorate Alzheimer's-like deficits in mice modeling the disease.
Chronic inhibition of mammalian target of rapamycin by rapamycin modulates cognitive and non-cognitive components of behavior throughout lifespan in mice.
The role of MAP4K3 in lifespan regulation of Caenorhabditis elegans.
Inhibition of mTOR by rapamycin abolishes cognitive deficits and reduces amyloid-beta levels in a mouse model of Alzheimer's disease.
An engineered transforming growth factor ß (TGF-ß) monomer that functions as a dominant negative to block TGF-ß signaling.
Vascular mTOR-dependent mechanisms linking the control of aging to Alzheimer's disease.
Protein Serine Threonine Kinases