"Cell Growth Processes" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Processes required for CELL ENLARGEMENT and CELL PROLIFERATION.
Below are MeSH descriptors whose meaning is more general than "Cell Growth Processes".
Below are MeSH descriptors whose meaning is more specific than "Cell Growth Processes".
This graph shows the total number of publications written about "Cell Growth Processes" by people in this website by year, and whether "Cell Growth Processes" was a major or minor topic of these publications.
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|Year||Major Topic||Minor Topic||Total|
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Below are the most recent publications written about "Cell Growth Processes" by people in Profiles.
G-CSF and G-CSFR are highly expressed in human gastric and colon cancers and promote carcinoma cell proliferation and migration. Br J Cancer. 2014 Mar 04; 110(5):1211-20.
SU11652 Inhibits tyrosine kinase activity of FLT3 and growth of MV-4-11 cells. J Hematol Oncol. 2012 Dec 06; 5:72.
Metformin potentiates the effects of paclitaxel in endometrial cancer cells through inhibition of cell proliferation and modulation of the mTOR pathway. Gynecol Oncol. 2012 May; 125(2):458-69.
p57 is required for quiescence and maintenance of adult hematopoietic stem cells. Cell Stem Cell. 2011 Sep 02; 9(3):262-71.
18a-glycyrrhetinic acid targets prostate cancer cells by down-regulating inflammation-related genes. Int J Oncol. 2011 Sep; 39(3):635-40.
Influence of novel KGFR tyrosine kinase inhibitors on KGF-mediated proliferation of breast cancer. Anticancer Res. 2010 Dec; 30(12):4883-9.
Novel STAT3 phosphorylation inhibitors exhibit potent growth-suppressive activity in pancreatic and breast cancer cells. Cancer Res. 2010 Mar 15; 70(6):2445-54.
MiR-15a and MiR-16 control Bmi-1 expression in ovarian cancer. Cancer Res. 2009 Dec 01; 69(23):9090-5.
Metformin is a potent inhibitor of endometrial cancer cell proliferation--implications for a novel treatment strategy. Gynecol Oncol. 2010 Jan; 116(1):92-8.
Suppression of familial adenomatous polyposis by CP-31398, a TP53 modulator, in APCmin/+ mice. Cancer Res. 2008 Sep 15; 68(18):7670-5.