Dual Specificity Phosphatase 6
"Dual Specificity Phosphatase 6" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A dual specificity phosphatase subtype that plays a role in intracellular signal transduction by inactivating MITOGEN-ACTIVATED PROTEIN KINASES. It has specificity for EXTRACELLULAR SIGNAL-REGULATED MAP KINASES and is primarily localized to the CYTOSOL.
| Descriptor ID |
D054642
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| MeSH Number(s) |
D08.811.277.352.650.587.600 D08.811.277.352.650.625.225.600 D08.811.277.352.650.775.250.600 D08.811.641.755.600 D12.644.360.268.600 D12.644.360.445.600 D12.776.476.268.600 D12.776.476.445.600
|
| Concept/Terms |
Dual Specificity Phosphatase 6- Dual Specificity Phosphatase 6
- Dual Specificity Protein Phosphatase 6
- MKP-3 Phosphatase
- MKP 3 Phosphatase
- Phosphatase, MKP-3
- MKP3 Phosphatase
- Phosphatase, MKP3
- MAP Kinase Phosphatase 3
- Mitogen-Activated Protein Kinase Phosphatase 3
- Mitogen Activated Protein Kinase Phosphatase 3
|
Below are MeSH descriptors whose meaning is more general than "Dual Specificity Phosphatase 6".
Below are MeSH descriptors whose meaning is more specific than "Dual Specificity Phosphatase 6".
This graph shows the total number of publications written about "Dual Specificity Phosphatase 6" by people in this website by year, and whether "Dual Specificity Phosphatase 6" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2006 | 0 | 1 | 1 |
| 2014 | 1 | 0 | 1 |
| 2015 | 0 | 1 | 1 |
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Below are the most recent publications written about "Dual Specificity Phosphatase 6" by people in Profiles.
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mir-181a-1/b-1 Modulates Tolerance through Opposing Activities in Selection and Peripheral T Cell Function. J Immunol. 2015 Aug 15; 195(4):1470-9.
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Mitogen-activated protein kinase phosphatase 3 (MKP-3)-deficient mice are resistant to diet-induced obesity. Diabetes. 2014 Sep; 63(9):2924-34.
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Multiple oncogenic changes (K-RAS(V12), p53 knockdown, mutant EGFRs, p16 bypass, telomerase) are not sufficient to confer a full malignant phenotype on human bronchial epithelial cells. Cancer Res. 2006 Feb 15; 66(4):2116-28.