"Mice, Inbred C57BL" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
One of the first INBRED MOUSE STRAINS to be sequenced. This strain is commonly used as genetic background for transgenic mouse models. Refractory to many tumors, this strain is also preferred model for studying role of genetic variations in development of diseases.
Descriptor ID |
D008810
|
MeSH Number(s) |
B01.050.050.199.520.520.420 B01.050.150.900.649.313.992.635.505.500.400.420
|
Concept/Terms |
Mice, Inbred C57BL- Mice, Inbred C57BL
- C57BL Mice, Inbred
- Inbred C57BL Mice
- Mouse, Inbred C57BL
- C57BL Mouse, Inbred
- Inbred C57BL Mouse
- Mice, C57BL
- C57BL Mice
- Mouse, C57BL
- C57BL Mouse
|
Below are MeSH descriptors whose meaning is more general than "Mice, Inbred C57BL".
Below are MeSH descriptors whose meaning is more specific than "Mice, Inbred C57BL".
This graph shows the total number of publications written about "Mice, Inbred C57BL" by people in this website by year, and whether "Mice, Inbred C57BL" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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1994 | 0 | 6 | 6 |
1995 | 0 | 5 | 5 |
1996 | 0 | 4 | 4 |
1997 | 0 | 6 | 6 |
1998 | 0 | 6 | 6 |
1999 | 0 | 6 | 6 |
2000 | 0 | 8 | 8 |
2001 | 0 | 5 | 5 |
2002 | 0 | 9 | 9 |
2003 | 0 | 12 | 12 |
2004 | 0 | 16 | 16 |
2005 | 0 | 19 | 19 |
2006 | 0 | 23 | 23 |
2007 | 0 | 26 | 26 |
2008 | 0 | 29 | 29 |
2009 | 0 | 28 | 28 |
2010 | 0 | 44 | 44 |
2011 | 0 | 41 | 41 |
2012 | 0 | 52 | 52 |
2013 | 0 | 47 | 47 |
2014 | 0 | 55 | 55 |
2015 | 0 | 42 | 42 |
2016 | 0 | 52 | 52 |
2017 | 0 | 52 | 52 |
2018 | 0 | 53 | 53 |
2019 | 0 | 50 | 50 |
2020 | 0 | 59 | 59 |
2021 | 0 | 57 | 57 |
2022 | 0 | 17 | 17 |
2023 | 0 | 10 | 10 |
2024 | 3 | 23 | 26 |
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Below are the most recent publications written about "Mice, Inbred C57BL" by people in Profiles.
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Gpx4 Regulates Invariant NKT Cell Homeostasis and Function by Preventing Lipid Peroxidation and Ferroptosis. J Immunol. 2024 Oct 01; 213(7):941-951.
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Promotion of endoplasmic reticulum retrotranslocation by overexpression of E3 ubiquitin-protein ligase synoviolin 1 reduces endoplasmic reticulum stress and preserves cone photoreceptors in cyclic nucleotide-gated channel deficiency. FASEB J. 2024 Sep 15; 38(17):e70021.
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Dysregulated Ca2+ signaling, fluid secretion, and mitochondrial function in a mouse model of early Sj?gren's disease. Elife. 2024 Sep 11; 13.
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Deficient RPE mitochondrial energetics leads to subretinal fibrosis in age-related neovascular macular degeneration. Commun Biol. 2024 Sep 02; 7(1):1075.
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Osteopontin deletion attenuates cyst growth but exacerbates fibrosis in mice with cystic kidney disease. Physiol Rep. 2024 Sep; 12(17):e70038.
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Acyl-CoA synthetase 6 controls rod photoreceptor function and survival by shaping the phospholipid composition of retinal membranes. Commun Biol. 2024 Aug 21; 7(1):1027.
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Age-related retinal degeneration resulting from the deletion of Shp2 tyrosine phosphatase in photoreceptor neurons. Cell Death Dis. 2024 Aug 08; 15(8):577.
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MASLD does not affect fertility and senolytics fail to prevent MASLD progression in male mice. Sci Rep. 2024 07 27; 14(1):17332.
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DHODH inhibition enhances the efficacy of immune checkpoint blockade by increasing cancer cell antigen presentation. Elife. 2024 Jul 08; 12.
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Transcriptomic Analysis Reveals That Excessive Thyroid Hormone Signaling Impairs Phototransduction and Mitochondrial Bioenergetics and Induces Cellular Stress in Mouse Cone Photoreceptors. Int J Mol Sci. 2024 Jul 06; 25(13).