beta-Adrenergic Receptor Kinases
"beta-Adrenergic Receptor Kinases" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
G-protein-coupled receptor kinases that mediate agonist-dependent PHOSPHORYLATION and desensitization of BETA-ADRENERGIC RECEPTORS.
| Descriptor ID |
D051552
|
| MeSH Number(s) |
D08.811.913.696.620.682.700.364.049 D12.644.360.293.249 D12.776.476.293.249
|
| Concept/Terms |
beta-Adrenergic Receptor Kinases- beta-Adrenergic Receptor Kinases
- Receptor Kinases, beta-Adrenergic
- beta Adrenergic Receptor Kinases
- beta-AR Kinase
- beta AR Kinase
- beta-Adrenergic Receptor Kinase
- Receptor Kinase, beta-Adrenergic
- beta Adrenergic Receptor Kinase
|
Below are MeSH descriptors whose meaning is more general than "beta-Adrenergic Receptor Kinases".
Below are MeSH descriptors whose meaning is more specific than "beta-Adrenergic Receptor Kinases".
This graph shows the total number of publications written about "beta-Adrenergic Receptor Kinases" by people in this website by year, and whether "beta-Adrenergic Receptor Kinases" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2002 | 0 | 3 | 3 |
| 2004 | 0 | 1 | 1 |
| 2005 | 0 | 1 | 1 |
| 2006 | 1 | 0 | 1 |
| 2007 | 1 | 0 | 1 |
To return to the timeline,
click here.
Below are the most recent publications written about "beta-Adrenergic Receptor Kinases" by people in Profiles.
-
Extracellular signal-regulated kinase 1/2-mediated transcriptional regulation of G-protein-coupled receptor kinase 3 expression in neuronal cells. J Pharmacol Exp Ther. 2007 Apr; 321(1):51-9.
-
Involvement of G protein-coupled receptor kinase (GRK) 3 and GRK2 in down-regulation of the alpha2B-adrenoceptor. J Pharmacol Exp Ther. 2006 Jun; 317(3):1027-35.
-
Tumor necrosis factor alpha, rapid ventricular tachyarrhythmias, and infarct size in canine models of myocardial infarction. J Cardiovasc Pharmacol. 2005 Feb; 45(2):153-9.
-
Cellular G protein-coupled receptor kinase levels regulate sensitivity of the {alpha}2b-adrenergic receptor to undergo agonist-induced down-regulation. J Pharmacol Exp Ther. 2005 Feb; 312(2):767-73.
-
Induction of G protein-coupled receptor kinases 2 and 3 contributes to the cross-talk between mu and ORL1 receptors following prolonged agonist exposure. Neuropharmacology. 2002 Nov; 43(6):979-90.
-
Orphanin FQ/nociceptin-mediated desensitization of opioid receptor-like 1 receptor and mu opioid receptors involves protein kinase C: a molecular mechanism for heterologous cross-talk. J Pharmacol Exp Ther. 2002 Aug; 302(2):502-9.
-
Regulation of inflammatory responses. Penn Dent J (Phila). 2002; 102:8-9.