Macrophage Inflammatory Proteins
"Macrophage Inflammatory Proteins" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Heparin-binding proteins that exhibit a number of inflammatory and immunoregulatory activities. Originally identified as secretory products of MACROPHAGES, these chemokines are produced by a variety of cell types including NEUTROPHILS; FIBROBLASTS; and EPITHELIAL CELLS. They likely play a significant role in respiratory tract defenses.
Descriptor ID |
D019402
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MeSH Number(s) |
D12.644.276.374.200.600 D12.776.467.374.200.600 D23.125.300.600 D23.469.200.600 D23.529.374.200.600
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Concept/Terms |
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Below are MeSH descriptors whose meaning is more general than "Macrophage Inflammatory Proteins".
Below are MeSH descriptors whose meaning is more specific than "Macrophage Inflammatory Proteins".
This graph shows the total number of publications written about "Macrophage Inflammatory Proteins" by people in this website by year, and whether "Macrophage Inflammatory Proteins" was a major or minor topic of these publications.
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Year | Major Topic | Minor Topic | Total |
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2004 | 0 | 1 | 1 |
2006 | 0 | 1 | 1 |
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Below are the most recent publications written about "Macrophage Inflammatory Proteins" by people in Profiles.
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Matrix metalloproteinase-9 and tissue inhibitor of matrix metalloproteinase-1 in the respiratory tracts of human infants following paramyxovirus infection. J Med Virol. 2007 Apr; 79(4):447-56.
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The lack of RNA-dependent protein kinase enhances susceptibility of mice to genital herpes simplex virus type 2 infection. Immunology. 2006 Aug; 118(4):520-6.
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A comparison of epidemiologic and immunologic features of bronchiolitis caused by influenza virus and respiratory syncytial virus. J Med Virol. 2005 Feb; 75(2):282-9.
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C3a enhances nerve growth factor-induced NFAT activation and chemokine production in a human mast cell line, HMC-1. J Immunol. 2004 Jun 01; 172(11):6961-8.
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IkappaB kinase is a critical regulator of chemokine expression and lung inflammation in respiratory syncytial virus infection. J Virol. 2004 Mar; 78(5):2232-41.
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Macrophage inflammatory protein-1alpha (not T helper type 2 cytokines) is associated with severe forms of respiratory syncytial virus bronchiolitis. J Infect Dis. 2001 Aug 15; 184(4):393-9.
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Inducible expression of inflammatory chemokines in respiratory syncytial virus-infected mice: role of MIP-1alpha in lung pathology. J Virol. 2001 Jan; 75(2):878-90.