"Inclusion Bodies" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A generic term for any circumscribed mass of foreign (e.g., lead or viruses) or metabolically inactive materials (e.g., ceroid or MALLORY BODIES), within the cytoplasm or nucleus of a cell. Inclusion bodies are in cells infected with certain filtrable viruses, observed especially in nerve, epithelial, or endothelial cells. (Stedman, 25th ed)
| Descriptor ID |
D002479
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| MeSH Number(s) |
A11.284.420
|
| Concept/Terms |
Inclusion Bodies- Inclusion Bodies
- Bodies, Inclusion
- Body, Inclusion
- Inclusion Body
- Cellular Inclusions
- Cellular Inclusion
- Inclusion, Cellular
- Inclusions, Cellular
Cytoplasmic Inclusions- Cytoplasmic Inclusions
- Cytoplasmic Inclusion
- Inclusion, Cytoplasmic
- Inclusions, Cytoplasmic
|
Below are MeSH descriptors whose meaning is more general than "Inclusion Bodies".
Below are MeSH descriptors whose meaning is more specific than "Inclusion Bodies".
This graph shows the total number of publications written about "Inclusion Bodies" by people in this website by year, and whether "Inclusion Bodies" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2010 | 1 | 0 | 1 |
| 2011 | 0 | 1 | 1 |
| 2012 | 0 | 1 | 1 |
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Below are the most recent publications written about "Inclusion Bodies" by people in Profiles.
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Genetic Inactivation of Chlamydia trachomatis Inclusion Membrane Protein CT228 Alters MYPT1 Recruitment, Extrusion Production, and Longevity of Infection. Front Cell Infect Microbiol. 2018; 8:415.
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Cigarette smoke-induced autophagy impairment accelerates lung aging, COPD-emphysema exacerbations and pathogenesis. Am J Physiol Cell Physiol. 2018 01 01; 314(1):C73-C87.
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CyanoPhyChe: a database for physico-chemical properties, structure and biochemical pathway information of cyanobacterial proteins. PLoS One. 2012; 7(11):e49425.
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The laforin-malin complex negatively regulates glycogen synthesis by modulating cellular glucose uptake via glucose transporters. Mol Cell Biol. 2012 Feb; 32(3):652-63.
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Prediction of protein solubility in Escherichia coli using logistic regression. Biotechnol Bioeng. 2010 Feb 01; 105(2):374-83.
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Impaired protein aggregate handling and clearance underlie the pathogenesis of p97/VCP-associated disease. J Biol Chem. 2008 Oct 31; 283(44):30289-99.
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Transgenic expression of inclusion body myopathy associated mutant p97/VCP causes weakness and ubiquitinated protein inclusions in mice. Hum Mol Genet. 2007 Apr 15; 16(8):919-28.
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Cytomegalovirus-associated cutaneous vasculopathy and scleroderma sans inclusion body change. Hum Pathol. 2007 Jan; 38(1):42-9.
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Immunoglobulin A-associated lymphocytic vasculopathy: a clinicopathologic study of eight patients. J Cutan Pathol. 2002 Nov; 29(10):596-601.
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Drug therapy of the idiopathic inflammatory myopathies: predictors of response to prednisone, azathioprine, and methotrexate and a comparison of their efficacy. Am J Med. 1993 Apr; 94(4):379-87.