A misspliced form of the cholecystokinin-B/gastrin receptor in pancreatic carcinoma: role of reduced sellular U2AF35 and a suboptimal 3'-splicing site leading to retention of the fourth intron.

A misspliced form of the cholecystokinin-B/gastrin receptor in pancreatic carcinoma: role of reduced sellular U2AF35 and a suboptimal 3'-splicing site leading to retention of the fourth intron. Cancer Res. 2002 Feb 01; 62(3):947-52.

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subject areas

Alternative Splicing
Amino Acid Sequence
Humans
Introns
Molecular Sequence Data
Nuclear Proteins
Pancreatic Neoplasms
Receptor, Cholecystokinin B
Receptors, Cholecystokinin
Ribonucleoproteins
RNA, Messenger
Splicing Factor U2AF
Tumor Cells, Cultured

authors with profiles

Wei-Qun Ding