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Aconitase regulation of erythropoiesis correlates with a novel licensing function in erythropoietin-induced ERK signaling.
Elevated levels of mast cells are involved in pruritus associated with polycythemia vera in JAK2V617F transgenic mice.
Erlotinib is not effective in patients with JAK2V617F-positive polycythemia vera.
Thrombopoietin is required for full phenotype expression in a JAK2V617F transgenic mouse model of polycythemia vera.
Role of tyrosine kinases and phosphatases in polycythemia vera.
Erlotinib effectively inhibits JAK2V617F activity and polycythemia vera cell growth.
PTP-MEG2 is activated in polycythemia vera erythroid progenitor cells and is required for growth and expansion of erythroid cells.
Identification of an acquired JAK2 mutation in polycythemia vera.
Detection of the JAK2V617F mutation by asymmetric PCR and melt curve analysis.
CD169? macrophages provide a niche promoting erythropoiesis under homeostasis and stress.
PROTEIN TYROSINE PHOSPHATASES IN POLYCYTHEMIA VERA
Tyrosine Phosphatase PTP-MEG2 and Hematopoiesis
Tyrosine Kinase JAK2 and Myeloproliferative Disorders